Hypertension-induced remodeling of cardiac excitation-contraction coupling in ventricular myocytes occurs prior to hypertrophy development

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 293, Issue 6, 2007, Pages

  Chen Izu, Ye ^a   Chen, Ling ^b   Bányász, Tamás ^a,c   McCulle, Stacey L ^b   Norton, Byron ^a   Scharf, Steven M ^b   Agarwal, Anuj ^d   Patwardhan, Abhijit ^d   Izu, Leighton T ^a   Balke, C William ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b UNIVERSITY OF MARYLAND SCHOOL OF MEDICINE   (United States)

^c UNIVERSITY OF DEBRECEN   (Hungary)

^d UNIVERSITY OF KENTUCKY   (United States)

Author keywords

Action potential; Heart failure; L type Ca2+ channel; Rryanodine receptor

Indexed keywords

CALCIUM ION;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DISEASE ASSOCIATION; DISEASE COURSE; ECHOCARDIOGRAPHY; EXCITATION CONTRACTION COUPLING; HEART CONTRACTION; HEART HEMODYNAMICS; HEART LEFT VENTRICLE EJECTION FRACTION; HEART MUSCLE CELL; HEART OUTPUT; HEART VENTRICLE HYPERTROPHY; HEART VENTRICLE REMODELING; HYPERTENSION; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; SPONTANEOUSLY HYPERTENSIVE RAT;

ACTION POTENTIALS; ANIMALS; CALCIUM SIGNALING; CARDIAC OUTPUT; CARDIOMEGALY; DISEASE MODELS, ANIMAL; DISEASE PROGRESSION; HEART VENTRICLES; HYPERTENSION; MALE; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; RATS; RATS, INBRED SHR; RATS, INBRED WKY; SARCOPLASMIC RETICULUM; TIME FACTORS; VENTRICULAR FUNCTION, LEFT; VENTRICULAR REMODELING;

EID: 36849068571     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00259.2007     Document Type: Article

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