-
1
-
-
9644268167
-
Insulin resistance contributes to aberrant stress responses in the Tg2576 mouse model of Alzheimer's disease
-
Pedersen, W. A., and E. R. Flynn. 2004. Insulin resistance contributes to aberrant stress responses in the Tg2576 mouse model of Alzheimer's disease. Neurobiol. Dis. 17: 500-506.
-
(2004)
Neurobiol. Dis
, vol.17
, pp. 500-506
-
-
Pedersen, W.A.1
Flynn, E.R.2
-
2
-
-
33745234766
-
Rosiglitazone attenuates learning and memory deficits in Tg2576 Alzheimer mice
-
Pedersen, W. A., P. J. McMillan, J. J. Kulstad, J. B. Leverenz, S. Craft, and G. R. Haynatzki. 2006. Rosiglitazone attenuates learning and memory deficits in Tg2576 Alzheimer mice. Exp. Neurol. 199: 265-273.
-
(2006)
Exp. Neurol
, vol.199
, pp. 265-273
-
-
Pedersen, W.A.1
McMillan, P.J.2
Kulstad, J.J.3
Leverenz, J.B.4
Craft, S.5
Haynatzki, G.R.6
-
3
-
-
20044392282
-
The R6/2 transgenic mouse model of Huntington's disease develops diabetes due to deficient beta-cell mass and exocytosis
-
Bjorkqvist, M., M. Fex, E. Renstrom, N. Wierup, A. Petersen, J. Gil, K. Bacos, N. Popovic, J. Y. Li, F. Sundler, et al. 2005. The R6/2 transgenic mouse model of Huntington's disease develops diabetes due to deficient beta-cell mass and exocytosis. Hum. Mol. Genet. 14: 565-574.
-
(2005)
Hum. Mol. Genet
, vol.14
, pp. 565-574
-
-
Bjorkqvist, M.1
Fex, M.2
Renstrom, E.3
Wierup, N.4
Petersen, A.5
Gil, J.6
Bacos, K.7
Popovic, N.8
Li, J.Y.9
Sundler, F.10
-
4
-
-
0035862874
-
Abnormalities in the functioning of adipocytes from R6/2 mice that are transgenic for the Huntington's disease mutation
-
Fain, J. N., N. A. Del Mar, C. A. Meade, A. Reiner, and D. Goldowitz. 2001. Abnormalities in the functioning of adipocytes from R6/2 mice that are transgenic for the Huntington's disease mutation. Hum. Mol. Genet. 10: 145-152.
-
(2001)
Hum. Mol. Genet
, vol.10
, pp. 145-152
-
-
Fain, J.N.1
Del Mar, N.A.2
Meade, C.A.3
Reiner, A.4
Goldowitz, D.5
-
5
-
-
0033009587
-
Mice transgenic for an expanded CAG repeat in the Huntington's disease gene develop diabetes
-
Hurlbert, M. S., W. Zhou, C. Wasmeier, F. G. Kaddis, J. C. Hutton, and C. R. Freed. 1999. Mice transgenic for an expanded CAG repeat in the Huntington's disease gene develop diabetes. Diabetes. 48: 649-651.
-
(1999)
Diabetes
, vol.48
, pp. 649-651
-
-
Hurlbert, M.S.1
Zhou, W.2
Wasmeier, C.3
Kaddis, F.G.4
Hutton, J.C.5
Freed, C.R.6
-
6
-
-
3343020674
-
Evidence for defective energy homeostasis in amyotrophic lateral sclerosis: Benefit of a high-energy diet in a transgenic mouse model
-
Dupuis, L., H. Oudart, F. Rene, J. L. Gonzalez de Aguilar, and J. P. Loeffler. 2004. Evidence for defective energy homeostasis in amyotrophic lateral sclerosis: benefit of a high-energy diet in a transgenic mouse model. Proc. Natl. Acad. Sci. USA. 101: 11159-11164.
-
(2004)
Proc. Natl. Acad. Sci. USA
, vol.101
, pp. 11159-11164
-
-
Dupuis, L.1
Oudart, H.2
Rene, F.3
Gonzalez de Aguilar, J.L.4
Loeffler, J.P.5
-
7
-
-
33750437278
-
Thermoregulatory and metabolic defects in Huntington's disease transgenic mice implicate PGC-1alpha in Huntington's disease neurodegeneration
-
Weydt, P., V. V. Pineda, A. E. Torrence, R. T. Libby, T. F. Satterfield, E. R. Lazarowski, M. L. Gilbert, G. J. Morton, T. K. Bammler, A. D. Strand, et al. 2006. Thermoregulatory and metabolic defects in Huntington's disease transgenic mice implicate PGC-1alpha in Huntington's disease neurodegeneration. Cell Metab. 4: 349-362.
-
(2006)
Cell Metab
, vol.4
, pp. 349-362
-
-
Weydt, P.1
Pineda, V.V.2
Torrence, A.E.3
Libby, R.T.4
Satterfield, T.F.5
Lazarowski, E.R.6
Gilbert, M.L.7
Morton, G.J.8
Bammler, T.K.9
Strand, A.D.10
-
8
-
-
33747605320
-
Molecular biology of amyotrophic lateral sclerosis: Insights from genetics
-
Pasinelli, P., and R. H. Brown. 2006. Molecular biology of amyotrophic lateral sclerosis: insights from genetics. Nat. Rev. Neurosci. 7: 710-723.
-
(2006)
Nat. Rev. Neurosci
, vol.7
, pp. 710-723
-
-
Pasinelli, P.1
Brown, R.H.2
-
9
-
-
34248149517
-
Amyotrophic lateral sclerosis: All roads lead to Rome
-
Gonzalez de Aguilar, J. L., A. Echaniz-Laguna, A. Fergani, F. René, V. Meininger, J. P. Loeffler, and L. Dupuis. 2007. Amyotrophic lateral sclerosis: all roads lead to Rome. J. Neurochem. 101: 1153-1160.
-
(2007)
J. Neurochem
, vol.101
, pp. 1153-1160
-
-
Gonzalez de Aguilar, J.L.1
Echaniz-Laguna, A.2
Fergani, A.3
René, F.4
Meininger, V.5
Loeffler, J.P.6
Dupuis, L.7
-
10
-
-
0028888945
-
Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis
-
Ripps, M. E., G. W. Huntley, P. R. Hof, J. H. Morrison, and J. W. Gordon. 1995. Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis. Proc. Natl. Acad. Sci. USA. 92: 689-693.
-
(1995)
Proc. Natl. Acad. Sci. USA
, vol.92
, pp. 689-693
-
-
Ripps, M.E.1
Huntley, G.W.2
Hof, P.R.3
Morrison, J.H.4
Gordon, J.W.5
-
11
-
-
0028284779
-
Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation
-
Gurney, M. E., H. Pu, A. Y. Chiu, M. C. Dal Canto, C. Y. Polchow, D. D. Alexander, J. Caliendo, A. Hentati, Y. W. Kwon, and H. X. Deng. 1994. Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation. Science. 264: 1772-1775.
-
(1994)
Science
, vol.264
, pp. 1772-1775
-
-
Gurney, M.E.1
Pu, H.2
Chiu, A.Y.3
Dal Canto, M.C.4
Polchow, C.Y.5
Alexander, D.D.6
Caliendo, J.7
Hentati, A.8
Kwon, Y.W.9
Deng, H.X.10
-
12
-
-
0029053881
-
An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria
-
Wong, P. C., C. A. Pardo, D. R. Borchelt, M. K. Lee, N. G. Copeland, N. A. Jenkins, S. S. Sisodia, D. W. Cleveland, and D. L. Price. 1995. An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria. Neuron. 14: 1105-1116.
-
(1995)
Neuron
, vol.14
, pp. 1105-1116
-
-
Wong, P.C.1
Pardo, C.A.2
Borchelt, D.R.3
Lee, M.K.4
Copeland, N.G.5
Jenkins, N.A.6
Sisodia, S.S.7
Cleveland, D.W.8
Price, D.L.9
-
13
-
-
0027401203
-
Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis
-
Rosen, D. R., T. Siddique, D. Patterson, D. A. Figlewicz, P. Sapp, A. Hentati, D. Donaldson, J. Goto, J. P. O'Regan, H. X. Deng, et al. 1993. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis. Nature. 362: 59-62.
-
(1993)
Nature
, vol.362
, pp. 59-62
-
-
Rosen, D.R.1
Siddique, T.2
Patterson, D.3
Figlewicz, D.A.4
Sapp, P.5
Hentati, A.6
Donaldson, D.7
Goto, J.8
O'Regan, J.P.9
Deng, H.X.10
-
14
-
-
0141642203
-
Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice
-
Clement, A. M., M. D. Nguyen, E. A. Roberts, M. L. Garcia, S. Boillee, M. Rule, A. P. McMahon, W. Doucette, D. Siwek, R. J. Ferrante, et al. 2003. Wild-type nonneuronal cells extend survival of SOD1 mutant motor neurons in ALS mice. Science. 302: 113-117.
-
(2003)
Science
, vol.302
, pp. 113-117
-
-
Clement, A.M.1
Nguyen, M.D.2
Roberts, E.A.3
Garcia, M.L.4
Boillee, S.5
Rule, M.6
McMahon, A.P.7
Doucette, W.8
Siwek, D.9
Ferrante, R.J.10
-
15
-
-
33744798774
-
Onset and progression in inherited ALS determined by motor neurons and microglia
-
Boillee, S., K. Yamanaka, C. S. Lobsiger, N. G. Copeland, N. A. Jenkins, G. Kassiotis, G. Kollias, and D. W. Cleveland. 2006. Onset and progression in inherited ALS determined by motor neurons and microglia. Science. 312: 1389-1392.
-
(2006)
Science
, vol.312
, pp. 1389-1392
-
-
Boillee, S.1
Yamanaka, K.2
Lobsiger, C.S.3
Copeland, N.G.4
Jenkins, N.A.5
Kassiotis, G.6
Kollias, G.7
Cleveland, D.W.8
-
16
-
-
0345426778
-
Up-regulation of mitochondrial uncoupling protein 3 reveals an early muscular metabolic defect in amyotrophic lateral sclerosis
-
Dupuis, L., F. di Scala, F. Rene, M. de Tapia, H. Oudart, P. F. Pradat, V. Meininger, and J. P. Loeffler. 2003. Up-regulation of mitochondrial uncoupling protein 3 reveals an early muscular metabolic defect in amyotrophic lateral sclerosis. FASEB J. 17: 2091-2093.
-
(2003)
FASEB J
, vol.17
, pp. 2091-2093
-
-
Dupuis, L.1
di Scala, F.2
Rene, F.3
de Tapia, M.4
Oudart, H.5
Pradat, P.F.6
Meininger, V.7
Loeffler, J.P.8
-
17
-
-
0034875640
-
Factors correlated with hypermetabolism in patients with amyotrophic lateral sclerosis
-
Desport, J. C., P. M. Preux, L. Magy, Y. Boirie, J. M. Vallat, B. Beaufrere, and P. Couratier. 2001. Factors correlated with hypermetabolism in patients with amyotrophic lateral sclerosis. Am. J. Clin. Nutr. 74: 328-334.
-
(2001)
Am. J. Clin. Nutr
, vol.74
, pp. 328-334
-
-
Desport, J.C.1
Preux, P.M.2
Magy, L.3
Boirie, Y.4
Vallat, J.M.5
Beaufrere, B.6
Couratier, P.7
-
18
-
-
0030042365
-
Nutritional status of patients with amyotrophic lateral sclerosis: Relation to the proximity of death
-
Kasarskis, E. J., S. Berryman, J. G. Vanderleest, A. R. Schneider, and C. J. McClain. 1996. Nutritional status of patients with amyotrophic lateral sclerosis: relation to the proximity of death. Am. J. Clin. Nutr. 63: 130-137.
-
(1996)
Am. J. Clin. Nutr
, vol.63
, pp. 130-137
-
-
Kasarskis, E.J.1
Berryman, S.2
Vanderleest, J.G.3
Schneider, A.R.4
McClain, C.J.5
-
19
-
-
18044366164
-
The metabolic hypothesis in amyotrophic lateral sclerosis: Insights from mutant Cu/Zn-superoxide dismutase mice
-
Gonzalez de Aguilar, J. L., L. Dupuis, H. Oudart, and J. P. Loeffler. 2005. The metabolic hypothesis in amyotrophic lateral sclerosis: insights from mutant Cu/Zn-superoxide dismutase mice. Biomed. Pharmacother. 59: 190-196.
-
(2005)
Biomed. Pharmacother
, vol.59
, pp. 190-196
-
-
Gonzalez de Aguilar, J.L.1
Dupuis, L.2
Oudart, H.3
Loeffler, J.P.4
-
21
-
-
0033606410
-
No benefit of dietary restriction on disease onset or progression in amyotrophic lateral sclerosis Cu/Zn-superoxide dismutase mutant mice
-
Pedersen, W. A., and M. P. Mattson. 1999. No benefit of dietary restriction on disease onset or progression in amyotrophic lateral sclerosis Cu/Zn-superoxide dismutase mutant mice. Brain Res. 833: 117-120.
-
(1999)
Brain Res
, vol.833
, pp. 117-120
-
-
Pedersen, W.A.1
Mattson, M.P.2
-
22
-
-
22744432789
-
Intestinal gluconeogenesis and glucose transport according to body fuel availability in rats
-
Habold, C., C. Foltzer-Jourdainne, Y. Le Maho, J. H. Lignot, and H. Oudart. 2005. Intestinal gluconeogenesis and glucose transport according to body fuel availability in rats. J. Physiol. 566: 575-586.
-
(2005)
J. Physiol
, vol.566
, pp. 575-586
-
-
Habold, C.1
Foltzer-Jourdainne, C.2
Le Maho, Y.3
Lignot, J.H.4
Oudart, H.5
-
24
-
-
0033486698
-
Gastrointestinal dysfunction in amyotrophic lateral sclerosis
-
Toepfer, M., C. Folwaczny, A. Klauser, R. L. Riepl, W. Muller-Felber, and D. Pongratz. 1999. Gastrointestinal dysfunction in amyotrophic lateral sclerosis. Amyotroph. Lateral Scler. Other Motor Neuron Disord. 1: 15-19.
-
(1999)
Amyotroph. Lateral Scler. Other Motor Neuron Disord
, vol.1
, pp. 15-19
-
-
Toepfer, M.1
Folwaczny, C.2
Klauser, A.3
Riepl, R.L.4
Muller-Felber, W.5
Pongratz, D.6
-
25
-
-
10044271037
-
SREBP transcription factors: Master regulators of lipid homeostasis
-
Eberle, D., B. Hegarty, P. Bossard, P. Ferre, and F. Foufelle. 2004. SREBP transcription factors: master regulators of lipid homeostasis. Biochimie. 86: 839-848.
-
(2004)
Biochimie
, vol.86
, pp. 839-848
-
-
Eberle, D.1
Hegarty, B.2
Bossard, P.3
Ferre, P.4
Foufelle, F.5
-
26
-
-
0037790917
-
The enzymes, regulation, and genetics of bile acid synthesis
-
Russell, D. W. 2003. The enzymes, regulation, and genetics of bile acid synthesis. Annu. Rev. Biochem. 72: 137-174.
-
(2003)
Annu. Rev. Biochem
, vol.72
, pp. 137-174
-
-
Russell, D.W.1
-
27
-
-
33744816556
-
Hypermetabolism in ALS: Correlations with clinical and paraclinical parameters
-
Desport, J. C., F. Torny, M. Lacoste, P. M. Preux, and P. Couratier. 2005. Hypermetabolism in ALS: correlations with clinical and paraclinical parameters. Neurodegener Dis. 2: 202-207.
-
(2005)
Neurodegener Dis
, vol.2
, pp. 202-207
-
-
Desport, J.C.1
Torny, F.2
Lacoste, M.3
Preux, P.M.4
Couratier, P.5
-
28
-
-
32644434546
-
Muscular mitochondrial function in amyotrophic lateral sclerosis is progressively altered as the disease develops: A temporal study in man
-
Echaniz-Laguna, A., J. Zoll, E. Ponsot, B. N'guessan, C. Tranchant, J. P. Loeffler, and E. Lampert. 2006. Muscular mitochondrial function in amyotrophic lateral sclerosis is progressively altered as the disease develops: a temporal study in man. Exp. Neurol. 198: 25-30.
-
(2006)
Exp. Neurol
, vol.198
, pp. 25-30
-
-
Echaniz-Laguna, A.1
Zoll, J.2
Ponsot, E.3
N'guessan, B.4
Tranchant, C.5
Loeffler, J.P.6
Lampert, E.7
-
29
-
-
0032716335
-
Visualization of defective mitochondrial function in skeletal muscle fibers of patients with sporadic amyotrophic lateral sclerosis
-
Vielhaber, S., K. Winkler, E. Kirches, D. Kunz, M. Buchner, H. Feistner, C. E. Elger, A. C. Ludolph, M. W. Riepe, and W. S. Kunz. 1999. Visualization of defective mitochondrial function in skeletal muscle fibers of patients with sporadic amyotrophic lateral sclerosis. J. Neurol. Sci. 169: 133-139.
-
(1999)
J. Neurol. Sci
, vol.169
, pp. 133-139
-
-
Vielhaber, S.1
Winkler, K.2
Kirches, E.3
Kunz, D.4
Buchner, M.5
Feistner, H.6
Elger, C.E.7
Ludolph, A.C.8
Riepe, M.W.9
Kunz, W.S.10
-
30
-
-
0033917463
-
Mitochondrial DNA abnormalities in skeletal muscle of patients with sporadic amyotrophic lateral sclerosis
-
Vielhaber, S., D. Kunz, K. Winkler, F. R. Wiedemann, E. Kirches, H. Feistner, H. J. Heinze, C. E. Elger, W. Schubert, and W. S. Kunz. 2000. Mitochondrial DNA abnormalities in skeletal muscle of patients with sporadic amyotrophic lateral sclerosis. Brain. 123: 1339-1348.
-
(2000)
Brain
, vol.123
, pp. 1339-1348
-
-
Vielhaber, S.1
Kunz, D.2
Winkler, K.3
Wiedemann, F.R.4
Kirches, E.5
Feistner, H.6
Heinze, H.J.7
Elger, C.E.8
Schubert, W.9
Kunz, W.S.10
-
31
-
-
23444445882
-
Mitochondrial changes in skeletal muscle in amyotrophic lateral sclerosis and other neurogenic atrophies
-
Krasnianski, A., M. Deschauer, S. Neudecker, F. N. Gellerich, T. Muller, B. G. Schoser, M. Krasnianski, and S. Zierz. 2005. Mitochondrial changes in skeletal muscle in amyotrophic lateral sclerosis and other neurogenic atrophies. Brain. 128: 1870-1876.
-
(2005)
Brain
, vol.128
, pp. 1870-1876
-
-
Krasnianski, A.1
Deschauer, M.2
Neudecker, S.3
Gellerich, F.N.4
Muller, T.5
Schoser, B.G.6
Krasnianski, M.7
Zierz, S.8
-
32
-
-
24944436389
-
Triglyceride-rich lipoprotein metabolism in unique VLDL receptor, LDL receptor, and LRP triple-deficient mice
-
Espirito Santo, S. M., P. C. Rensen, J. R. Goudriaan, A. Bensadoun, N. Bovenschen, P. J. Voshol, L. M. Havekes, and B. J. van Vlijmen. 2005. Triglyceride-rich lipoprotein metabolism in unique VLDL receptor, LDL receptor, and LRP triple-deficient mice. J. Lipid Res. 46: 1097-1102.
-
(2005)
J. Lipid Res
, vol.46
, pp. 1097-1102
-
-
Espirito Santo, S.M.1
Rensen, P.C.2
Goudriaan, J.R.3
Bensadoun, A.4
Bovenschen, N.5
Voshol, P.J.6
Havekes, L.M.7
van Vlijmen, B.J.8
-
33
-
-
3442880397
-
The VLDL receptor plays a major role in chylomicron metabolism by enhancing LPL-mediated triglyceride hydrolysis
-
Goudriaan, J. R., S. M. Espirito Santo, P. J. Voshol, B. Teusink, K. W. van Dijk, B. J. van Vlijmen, J. A. Romijn, L. M. Havekes, and P. C. Rensen. 2004. The VLDL receptor plays a major role in chylomicron metabolism by enhancing LPL-mediated triglyceride hydrolysis. J. Lipid Res. 45: 1475-1481.
-
(2004)
J. Lipid Res
, vol.45
, pp. 1475-1481
-
-
Goudriaan, J.R.1
Espirito Santo, S.M.2
Voshol, P.J.3
Teusink, B.4
van Dijk, K.W.5
van Vlijmen, B.J.6
Romijn, J.A.7
Havekes, L.M.8
Rensen, P.C.9
-
34
-
-
0037155899
-
Very low density lipoprotein (VLDL) receptor-deficient mice have reduced lipoprotein lipase activity. Possible causes of hypertriglyceridemia and reduced body mass with VLDL receptor deficiency
-
Yagyu, H., E. P. Lutz, Y. Kako, S. Marks, Y. Hu, S. Y. Choi, A. Bensadoun, and I. J. Goldberg. 2002. Very low density lipoprotein (VLDL) receptor-deficient mice have reduced lipoprotein lipase activity. Possible causes of hypertriglyceridemia and reduced body mass with VLDL receptor deficiency. J. Biol. Chem. 277: 10037-10043.
-
(2002)
J. Biol. Chem
, vol.277
, pp. 10037-10043
-
-
Yagyu, H.1
Lutz, E.P.2
Kako, Y.3
Marks, S.4
Hu, Y.5
Choi, S.Y.6
Bensadoun, A.7
Goldberg, I.J.8
-
35
-
-
18244393432
-
CD36 deficiency impairs intestinal lipid secretion and clearance of chylomicrons from the blood
-
Drover, V. A., M. Ajmal, F. Nassir, N. O. Davidson, A. M. Nauli, D. Sahoo, P. Tso, and N. A. Abumrad. 2005. CD36 deficiency impairs intestinal lipid secretion and clearance of chylomicrons from the blood. J. Clin. Invest. 115: 1290-1297.
-
(2005)
J. Clin. Invest
, vol.115
, pp. 1290-1297
-
-
Drover, V.A.1
Ajmal, M.2
Nassir, F.3
Davidson, N.O.4
Nauli, A.M.5
Sahoo, D.6
Tso, P.7
Abumrad, N.A.8
-
36
-
-
0033578604
-
Muscle-specific overexpression of FAT/CD36 enhances fatty acid oxidation by contracting muscle, reduces plasma triglycerides and fatty acids, and increases plasma glucose and insulin
-
Ibrahimi, A., A. Bonen, W. D. Blinn, T. Hajri, X. Li, K. Zhong, R. Cameron, and N. A. Abumrad. 1999. Muscle-specific overexpression of FAT/CD36 enhances fatty acid oxidation by contracting muscle, reduces plasma triglycerides and fatty acids, and increases plasma glucose and insulin. J. Biol. Chem. 274: 26761-26766.
-
(1999)
J. Biol. Chem
, vol.274
, pp. 26761-26766
-
-
Ibrahimi, A.1
Bonen, A.2
Blinn, W.D.3
Hajri, T.4
Li, X.5
Zhong, K.6
Cameron, R.7
Abumrad, N.A.8
-
37
-
-
33845876647
-
Gene transfer demonstrates that muscle is not a primary target for non-cell autonomous toxicity in familial amyotrophic lateral sclerosis
-
Miller, T. M., S. H. Kim, K. Yamanaka, M. Hester, P. Umapathi, H. Arnson, L. Rizo, J. R. Mendell, F. H. Gage, D. W. Cleveland, et al. 2006. Gene transfer demonstrates that muscle is not a primary target for non-cell autonomous toxicity in familial amyotrophic lateral sclerosis. Proc. Natl. Acad. Sci. USA. 103: 19546-19551.
-
(2006)
Proc. Natl. Acad. Sci. USA
, vol.103
, pp. 19546-19551
-
-
Miller, T.M.1
Kim, S.H.2
Yamanaka, K.3
Hester, M.4
Umapathi, P.5
Arnson, H.6
Rizo, L.7
Mendell, J.R.8
Gage, F.H.9
Cleveland, D.W.10
-
38
-
-
19944427435
-
Caloric restriction increases neurotrophic factor levels and attenuates neurochemical and behavioral deficits in a primate model of Parkinson's disease
-
Maswood, N., J. Young, E. Tilmont, Z. Zhang, D. M. Gash, G. A. Gerhardt, R. Grondin, G. S. Roth, J. Mattison, M. A. Lane, et al. 2004. Caloric restriction increases neurotrophic factor levels and attenuates neurochemical and behavioral deficits in a primate model of Parkinson's disease. Proc. Natl. Acad. Sci. USA. 101: 18171-18176.
-
(2004)
Proc. Natl. Acad. Sci. USA
, vol.101
, pp. 18171-18176
-
-
Maswood, N.1
Young, J.2
Tilmont, E.3
Zhang, Z.4
Gash, D.M.5
Gerhardt, G.A.6
Grondin, R.7
Roth, G.S.8
Mattison, J.9
Lane, M.A.10
-
39
-
-
0037418339
-
Dietary restriction normalizes glucose metabolism and BDNF levels, slows disease progression, and increases survival in huntingtin mutant mice
-
Duan, W., Z. Guo, H. Jiang, M. Ware, X. J. Li, and M. P. Mattson. 2003. Dietary restriction normalizes glucose metabolism and BDNF levels, slows disease progression, and increases survival in huntingtin mutant mice. Proc. Natl. Acad. Sci. USA. 100: 2911-2916.
-
(2003)
Proc. Natl. Acad. Sci. USA
, vol.100
, pp. 2911-2916
-
-
Duan, W.1
Guo, Z.2
Jiang, H.3
Ware, M.4
Li, X.J.5
Mattson, M.P.6
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