-
1
-
-
0041919374
-
Zinc metalloproteinase, ZMPSTE24, is mutated in mandibuloacral dysplasia
-
Agarwal A.K., Fryns J.P., Auchus R.J., and Garg A. Zinc metalloproteinase, ZMPSTE24, is mutated in mandibuloacral dysplasia. Hum Mol Genet 12 (2003) 1995-2001
-
(2003)
Hum Mol Genet
, vol.12
, pp. 1995-2001
-
-
Agarwal, A.K.1
Fryns, J.P.2
Auchus, R.J.3
Garg, A.4
-
2
-
-
0036829085
-
Down-regulation of p300/CBP histone acetyltransferase activates a senescence checkpoint in human melanocytes
-
Bandyopadhyay D., Okan N.A., Bales E., Nascimiento L., Cole P.A., and Medrano E.E. Down-regulation of p300/CBP histone acetyltransferase activates a senescence checkpoint in human melanocytes. Cancer Res 62 (2002) 6231-6239
-
(2002)
Cancer Res
, vol.62
, pp. 6231-6239
-
-
Bandyopadhyay, D.1
Okan, N.A.2
Bales, E.3
Nascimiento, L.4
Cole, P.A.5
Medrano, E.E.6
-
3
-
-
0036791026
-
Zmpste24 deficiency in mice causes spontaneous bone fracture, muscle weakness, and prelamin A processing defect
-
Bergo M.O., Gavino B., Ross J., Schmidt W.K., Hong C., Kendall L.V., et al. Zmpste24 deficiency in mice causes spontaneous bone fracture, muscle weakness, and prelamin A processing defect. Proc Nat Acad Sci USA 99 (2002) 13049-13054
-
(2002)
Proc Nat Acad Sci USA
, vol.99
, pp. 13049-13054
-
-
Bergo, M.O.1
Gavino, B.2
Ross, J.3
Schmidt, W.K.4
Hong, C.5
Kendall, L.V.6
-
4
-
-
33745915850
-
Nuclear lamins: laminopathies and their role in premature ageing
-
Broers J.L.V., Ramaekers F.C.S., Bonne G., Ben Yaou R., and Hutchison C.J. Nuclear lamins: laminopathies and their role in premature ageing. Physiol Rev 86 (2006) 967-1008
-
(2006)
Physiol Rev
, vol.86
, pp. 967-1008
-
-
Broers, J.L.V.1
Ramaekers, F.C.S.2
Bonne, G.3
Ben Yaou, R.4
Hutchison, C.J.5
-
5
-
-
20444449733
-
Altered pre-lamin A processing is a common mechanism leading to lipodystrophy
-
Capanni C., Mattioli E., Columbaro M., Lucarelli E., Parnaik V.K., Novelli G., et al. Altered pre-lamin A processing is a common mechanism leading to lipodystrophy. Hum Mol Genet 14 (2005) 1489-1502
-
(2005)
Hum Mol Genet
, vol.14
, pp. 1489-1502
-
-
Capanni, C.1
Mattioli, E.2
Columbaro, M.3
Lucarelli, E.4
Parnaik, V.K.5
Novelli, G.6
-
6
-
-
24644520772
-
Inhibiting farnesylation of progerin prevents the characteristic nuclear blebbing of Hutchinson-Gilford progeria syndrome
-
Capell B.C., Erdos M.R., Madigan J.P., Fiordalisi J.J., Varga R., Conneely K.N., et al. Inhibiting farnesylation of progerin prevents the characteristic nuclear blebbing of Hutchinson-Gilford progeria syndrome. Proc Nat Acad Sci USA 102 (2005) 12879-12884
-
(2005)
Proc Nat Acad Sci USA
, vol.102
, pp. 12879-12884
-
-
Capell, B.C.1
Erdos, M.R.2
Madigan, J.P.3
Fiordalisi, J.J.4
Varga, R.5
Conneely, K.N.6
-
7
-
-
0035146907
-
Transcriptional repression, apoptosis, human disease and the functional evolution of the nuclear lamina
-
Cohen M., Lee K.K., Wilson K.L., and Gruenbaum Y. Transcriptional repression, apoptosis, human disease and the functional evolution of the nuclear lamina. Trends Biochem Sci 26 (2001) 41-47
-
(2001)
Trends Biochem Sci
, vol.26
, pp. 41-47
-
-
Cohen, M.1
Lee, K.K.2
Wilson, K.L.3
Gruenbaum, Y.4
-
8
-
-
28344445866
-
Rescue of heterochromatin organization in Hutchinson-Gilford progeria by drug treatment
-
Columbaro M., Capanni C., Mattioli E., Novelli G., Parnaik V.K., Squarzoni S., et al. Rescue of heterochromatin organization in Hutchinson-Gilford progeria by drug treatment. Cell Mol Life Sci 62 (2005) 2669-2678
-
(2005)
Cell Mol Life Sci
, vol.62
, pp. 2669-2678
-
-
Columbaro, M.1
Capanni, C.2
Mattioli, E.3
Novelli, G.4
Parnaik, V.K.5
Squarzoni, S.6
-
9
-
-
17144398001
-
Prelamin A endoproteolytic processing in vitro by recombinant Zmpste24
-
Corrigan D.P., Kuszczak D., Rusiñol A.E., Thewke D.P., Hrycyna C.A., Michaelis S., et al. Prelamin A endoproteolytic processing in vitro by recombinant Zmpste24. Biochem J 387 (2005) 129-138
-
(2005)
Biochem J
, vol.387
, pp. 129-138
-
-
Corrigan, D.P.1
Kuszczak, D.2
Rusiñol, A.E.3
Thewke, D.P.4
Hrycyna, C.A.5
Michaelis, S.6
-
10
-
-
33745904741
-
Distinct structural and mechanical properties of the nuclear lamina in Hutchinson-Gilford progeria syndrome
-
Dahl K.N., Scaffidi P., Islam M.F., Yodh A.G., Wilson K.L., and Misteli T. Distinct structural and mechanical properties of the nuclear lamina in Hutchinson-Gilford progeria syndrome. Proc Nat Acad Sci USA 103 (2006) 10271-10276
-
(2006)
Proc Nat Acad Sci USA
, vol.103
, pp. 10271-10276
-
-
Dahl, K.N.1
Scaffidi, P.2
Islam, M.F.3
Yodh, A.G.4
Wilson, K.L.5
Misteli, T.6
-
11
-
-
33645116709
-
The truncated prelamin A in Hutchinson-Gilford progeria syndrome alters segregation of A-type and B-type lamin homopolymers
-
Delbarre E., Tramier M., Coppey-Moisan M., Gaillard C., Courvalin J.C., and Buendia B. The truncated prelamin A in Hutchinson-Gilford progeria syndrome alters segregation of A-type and B-type lamin homopolymers. Hum Mol Genet 15 (2006) 1113-1122
-
(2006)
Hum Mol Genet
, vol.15
, pp. 1113-1122
-
-
Delbarre, E.1
Tramier, M.2
Coppey-Moisan, M.3
Gaillard, C.4
Courvalin, J.C.5
Buendia, B.6
-
12
-
-
33645743546
-
Lamina-associated polypeptide 2α regulates cell cycle progression and differentiation via the retinoblastoma-E2F pathway
-
Dorner D., Vlcek S., Foeger N., Gajewski A., Makolm C., Gotzmann J., et al. Lamina-associated polypeptide 2α regulates cell cycle progression and differentiation via the retinoblastoma-E2F pathway. J Cell Biol 173 (2006) 83-93
-
(2006)
J Cell Biol
, vol.173
, pp. 83-93
-
-
Dorner, D.1
Vlcek, S.2
Foeger, N.3
Gajewski, A.4
Makolm, C.5
Gotzmann, J.6
-
13
-
-
0037673950
-
Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome
-
Eriksson M., Brown W.T., Gordon L.B., Glynn M.W., Singer J., Scott L., et al. Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome. Nature 423 (2003) 293-298
-
(2003)
Nature
, vol.423
, pp. 293-298
-
-
Eriksson, M.1
Brown, W.T.2
Gordon, L.B.3
Glynn, M.W.4
Singer, J.5
Scott, L.6
-
14
-
-
28344440157
-
Alterations of nuclear envelope and chromatin organization in mandibuloacral dysplasia, a rare form of laminopathy
-
Filesi I., Gullotta F., Lattanzi G., D'Apice M.R., Capanni C., Nardone A.M., et al. Alterations of nuclear envelope and chromatin organization in mandibuloacral dysplasia, a rare form of laminopathy. Physiol Genom 23 (2005) 150-158
-
(2005)
Physiol Genom
, vol.23
, pp. 150-158
-
-
Filesi, I.1
Gullotta, F.2
Lattanzi, G.3
D'Apice, M.R.4
Capanni, C.5
Nardone, A.M.6
-
15
-
-
33645060977
-
A protein farnesyltransferase inhibitor ameliorates disease in mouse model of progeria
-
Fong L.G., Frost D., Meta M., Qiao X., Yang S.H., Coffinier C., et al. A protein farnesyltransferase inhibitor ameliorates disease in mouse model of progeria. Science 311 (2006) 1621-1623
-
(2006)
Science
, vol.311
, pp. 1621-1623
-
-
Fong, L.G.1
Frost, D.2
Meta, M.3
Qiao, X.4
Yang, S.H.5
Coffinier, C.6
-
16
-
-
19944428509
-
Heterozygosity for Lmna deficiency eliminates the progeria-like phenotypes in Zmpste24-deficient mice
-
Fong L.G., Ng J.K., Meta M., Coté N., Yang S.H., Stewart C.L., et al. Heterozygosity for Lmna deficiency eliminates the progeria-like phenotypes in Zmpste24-deficient mice. Proc Nat Acad Sci USA 101 (2004) 18111-18116
-
(2004)
Proc Nat Acad Sci USA
, vol.101
, pp. 18111-18116
-
-
Fong, L.G.1
Ng, J.K.2
Meta, M.3
Coté, N.4
Yang, S.H.5
Stewart, C.L.6
-
17
-
-
27544498316
-
Incomplete processing of mutant lamin A in Hutchinson-Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition
-
Glynn M.W., and Glover T.W. Incomplete processing of mutant lamin A in Hutchinson-Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition. Hum Mol Genet 14 (2005) 2959-2969
-
(2005)
Hum Mol Genet
, vol.14
, pp. 2959-2969
-
-
Glynn, M.W.1
Glover, T.W.2
-
18
-
-
2942643923
-
Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson-Gilford progeria syndrome
-
Goldman R.D., Shumaker D.K., Erdos M.R., Eriksson M., Goldman A.E., Gordon L.B., et al. Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson-Gilford progeria syndrome. Proc Nat Acad Sci USA 101 (2004) 8963-8968
-
(2004)
Proc Nat Acad Sci USA
, vol.101
, pp. 8963-8968
-
-
Goldman, R.D.1
Shumaker, D.K.2
Erdos, M.R.3
Eriksson, M.4
Goldman, A.E.5
Gordon, L.B.6
-
19
-
-
30044434950
-
A-type lamin complexes and regenerative potential: a step towards understanding laminopathic diseases?
-
Gotzmann J., and Foisner R. A-type lamin complexes and regenerative potential: a step towards understanding laminopathic diseases?. Histochem Cell Biol 125 (2005) 33-41
-
(2005)
Histochem Cell Biol
, vol.125
, pp. 33-41
-
-
Gotzmann, J.1
Foisner, R.2
-
20
-
-
20944446928
-
LMNA mutation position predicts organ system involvement in laminopathies
-
Hegele R. LMNA mutation position predicts organ system involvement in laminopathies. Clin Genet 68 (2005) 31-34
-
(2005)
Clin Genet
, vol.68
, pp. 31-34
-
-
Hegele, R.1
-
21
-
-
0033401971
-
Colocalization of intranuclear lamin foci with RNA splicing factors
-
Jagatheesan G., Thanumalayan S., Muralikrishna B., Rangaraj N., Karande A.A., and Parnaik V.K. Colocalization of intranuclear lamin foci with RNA splicing factors. J Cell Sci 112 (1999) 4651-4661
-
(1999)
J Cell Sci
, vol.112
, pp. 4651-4661
-
-
Jagatheesan, G.1
Thanumalayan, S.2
Muralikrishna, B.3
Rangaraj, N.4
Karande, A.A.5
Parnaik, V.K.6
-
22
-
-
18444382032
-
The Ig-like structure of the C-terminal domain of lamin A/C, mutated in muscular dystrophies, cardiomyopathy, and partial lipodystrophy
-
Krimm I., Ostlund C., Gilquin B., Couprie J., Hossenlopp P., Mornon J.P., et al. The Ig-like structure of the C-terminal domain of lamin A/C, mutated in muscular dystrophies, cardiomyopathy, and partial lipodystrophy. Structure 10 (2002) 811-823
-
(2002)
Structure
, vol.10
, pp. 811-823
-
-
Krimm, I.1
Ostlund, C.2
Gilquin, B.3
Couprie, J.4
Hossenlopp, P.5
Mornon, J.P.6
-
23
-
-
33644765799
-
Cell biology: ageing nucleus gets out of shape
-
Lans H., and Hoeijmakers J.H. Cell biology: ageing nucleus gets out of shape. Nature 440 (2006) 32-34
-
(2006)
Nature
, vol.440
, pp. 32-34
-
-
Lans, H.1
Hoeijmakers, J.H.2
-
24
-
-
22544466685
-
Genomic instability in laminopathy-based premature aging
-
Liu B., Wang J., Chan K.M., Tjia W.M., Deng W., Guan X., et al. Genomic instability in laminopathy-based premature aging. Nat Med 11 (2005) 780-785
-
(2005)
Nat Med
, vol.11
, pp. 780-785
-
-
Liu, B.1
Wang, J.2
Chan, K.M.3
Tjia, W.M.4
Deng, W.5
Guan, X.6
-
25
-
-
26444463068
-
Inhibiting farnesylation reverses the nuclear morphology defect in a HeLa cell model for Hutchinson-Gilford progeria syndrome
-
Mallampalli M.P., Huyer G., Bendale P., Gelb M.H., and Michaelis S. Inhibiting farnesylation reverses the nuclear morphology defect in a HeLa cell model for Hutchinson-Gilford progeria syndrome. Proc Nat Acad Sci USA 102 (2005) 14416-14421
-
(2005)
Proc Nat Acad Sci USA
, vol.102
, pp. 14416-14421
-
-
Mallampalli, M.P.1
Huyer, G.2
Bendale, P.3
Gelb, M.H.4
Michaelis, S.5
-
26
-
-
16344392142
-
Laminopathies: involvement of structural proteins in the pathogenesis of an increasing number of human diseases
-
Maraldi N.M., Squarzoni S., Sabatelli P., Capanni C., Mattioli E., Ognibene A., et al. Laminopathies: involvement of structural proteins in the pathogenesis of an increasing number of human diseases. J Cell Physiol 203 (2005) 319-327
-
(2005)
J Cell Physiol
, vol.203
, pp. 319-327
-
-
Maraldi, N.M.1
Squarzoni, S.2
Sabatelli, P.3
Capanni, C.4
Mattioli, E.5
Ognibene, A.6
-
27
-
-
33744999431
-
Nuclear envelope proteins and chromatin arrangement: a pathogenic mechanism for laminopathies
-
Maraldi N.M., Lattanzi G., Capanni C., Columbaro M., Merlini L., Mattioli E., et al. Nuclear envelope proteins and chromatin arrangement: a pathogenic mechanism for laminopathies. Eur J Histochem 50 (2006) 1-8
-
(2006)
Eur J Histochem
, vol.50
, pp. 1-8
-
-
Maraldi, N.M.1
Lattanzi, G.2
Capanni, C.3
Columbaro, M.4
Merlini, L.5
Mattioli, E.6
-
28
-
-
33748134758
-
Laminopathies: a chromatin affair
-
Maraldi N.M., Lattanzi G., Capanni C., Columbaro M., Mattioli E., Sabatelli P., et al. Laminopathies: a chromatin affair. Adv Enzyme Regul 46 (2006) 33-49
-
(2006)
Adv Enzyme Regul
, vol.46
, pp. 33-49
-
-
Maraldi, N.M.1
Lattanzi, G.2
Capanni, C.3
Columbaro, M.4
Mattioli, E.5
Sabatelli, P.6
-
29
-
-
16344380741
-
Sequestration of pRb by cyclin D3 causes intranuclear reorganization of lamin A/C during muscle cell differentiation
-
Mariappan I., and Parnaik V.K. Sequestration of pRb by cyclin D3 causes intranuclear reorganization of lamin A/C during muscle cell differentiation. Mol Biol Cell 16 (2005) 1948-1960
-
(2005)
Mol Biol Cell
, vol.16
, pp. 1948-1960
-
-
Mariappan, I.1
Parnaik, V.K.2
-
30
-
-
1842854443
-
Lamin A/C binding protein LAP2alpha is required for nuclear anchorage of retinoblastoma protein
-
Markiewicz E., Dechat T., Foisner R., Quinlan R.A., and Hutchison C.J. Lamin A/C binding protein LAP2alpha is required for nuclear anchorage of retinoblastoma protein. Mol Biol Cell 13 (2002) 4401-4413
-
(2002)
Mol Biol Cell
, vol.13
, pp. 4401-4413
-
-
Markiewicz, E.1
Dechat, T.2
Foisner, R.3
Quinlan, R.A.4
Hutchison, C.J.5
-
31
-
-
0037673940
-
A progeroid syndrome in mice is caused by defects in A-type lamins
-
Mounkes L.C., Kozlov S., Hernandez L., Sullivan T., and Stewart C.L. A progeroid syndrome in mice is caused by defects in A-type lamins. Nature 423 (2003) 298-301
-
(2003)
Nature
, vol.423
, pp. 298-301
-
-
Mounkes, L.C.1
Kozlov, S.2
Hernandez, L.3
Sullivan, T.4
Stewart, C.L.5
-
32
-
-
2342458205
-
Aging and nuclear organization: lamins and progeria
-
Mounkes L.C., and Stewart C.L. Aging and nuclear organization: lamins and progeria. Curr Opin Cell Biol 16 (2004) 322-327
-
(2004)
Curr Opin Cell Biol
, vol.16
, pp. 322-327
-
-
Mounkes, L.C.1
Stewart, C.L.2
-
33
-
-
19544374472
-
Lamin A and ZMPSTE24 (FACE-1) defects cause nuclear disorganization and identify restrictive dermopathy as a lethal neonatal laminopathy
-
Navarro C.L., De Sandre-Giovannoli A., Bernard R., Boccaccio I., Boyer A., Genevieve D., et al. Lamin A and ZMPSTE24 (FACE-1) defects cause nuclear disorganization and identify restrictive dermopathy as a lethal neonatal laminopathy. Hum Mol Genet 13 (2004) 2493-2503
-
(2004)
Hum Mol Genet
, vol.13
, pp. 2493-2503
-
-
Navarro, C.L.1
De Sandre-Giovannoli, A.2
Bernard, R.3
Boccaccio, I.4
Boyer, A.5
Genevieve, D.6
-
34
-
-
20444506041
-
Loss of ZMPSTE24 (FACE-1) causes autosomal recessive restrictive dermopathy and accumulation of Lamin A precursors
-
Navarro C.L., Cadinanos J., De Sandre-Giovannoli A., Bernard R., Courrier S., Boccaccio I., et al. Loss of ZMPSTE24 (FACE-1) causes autosomal recessive restrictive dermopathy and accumulation of Lamin A precursors. Hum Mol Genet 14 (2005) 1503-1513
-
(2005)
Hum Mol Genet
, vol.14
, pp. 1503-1513
-
-
Navarro, C.L.1
Cadinanos, J.2
De Sandre-Giovannoli, A.3
Bernard, R.4
Courrier, S.5
Boccaccio, I.6
-
35
-
-
0036578920
-
Defective prelamin A processing and muscular and adipocyte alterations in Zmpste24 metalloproteinase-deficient mice
-
Pendas A.M., Zhou Z., Cadinanos J., Freije J.M., Wang J., Hultenby K., et al. Defective prelamin A processing and muscular and adipocyte alterations in Zmpste24 metalloproteinase-deficient mice. Nat Genet 31 (2002) 94-99
-
(2002)
Nat Genet
, vol.31
, pp. 94-99
-
-
Pendas, A.M.1
Zhou, Z.2
Cadinanos, J.3
Freije, J.M.4
Wang, J.5
Hultenby, K.6
-
36
-
-
0347955358
-
Histone methyltransferases direct different degrees of methylation to define distinct chromatin domains
-
Rice J.C., Briggs S.D., Ueberheide B., Barber C.M., Shabanowitz J., Hunt D.F., et al. Histone methyltransferases direct different degrees of methylation to define distinct chromatin domains. Mol Cell 12 (2003) 1591-1598
-
(2003)
Mol Cell
, vol.12
, pp. 1591-1598
-
-
Rice, J.C.1
Briggs, S.D.2
Ueberheide, B.3
Barber, C.M.4
Shabanowitz, J.5
Hunt, D.F.6
-
37
-
-
33748760066
-
Farnesylated lamins, progeroid syndromes and farnesyl transferase inhibitors
-
Rusiñol A.E., and Sinensky M.S. Farnesylated lamins, progeroid syndromes and farnesyl transferase inhibitors. J Cell Sci 119 (2006) 3265-3272
-
(2006)
J Cell Sci
, vol.119
, pp. 3265-3272
-
-
Rusiñol, A.E.1
Sinensky, M.S.2
-
38
-
-
17644373758
-
Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome
-
Scaffidi P., and Misteli T. Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome. Nat Med 11 (2005) 440-445
-
(2005)
Nat Med
, vol.11
, pp. 440-445
-
-
Scaffidi, P.1
Misteli, T.2
-
39
-
-
30844469352
-
Compound heterozygous ZMPSTE24 mutations reduce prelamin A processing and result in a severe progeroid phenotype
-
Shackleton S., Smallwood D.T., Clayton P., Wilson L.C., Agarwal A.K., Garg A., et al. Compound heterozygous ZMPSTE24 mutations reduce prelamin A processing and result in a severe progeroid phenotype. J Med Genet 42 (2005) e36
-
(2005)
J Med Genet
, vol.42
-
-
Shackleton, S.1
Smallwood, D.T.2
Clayton, P.3
Wilson, L.C.4
Agarwal, A.K.5
Garg, A.6
-
40
-
-
5144220584
-
Crystal structure of the human lamin A coil 2B dimmer: implications for the head-to-tail association of nuclear lamins
-
Strelkov S.V., Schumacher J., Burkhard P., Aebi U., and Herrmann H. Crystal structure of the human lamin A coil 2B dimmer: implications for the head-to-tail association of nuclear lamins. J Mol Biol 343 (2004) 1067-1080
-
(2004)
J Mol Biol
, vol.343
, pp. 1067-1080
-
-
Strelkov, S.V.1
Schumacher, J.2
Burkhard, P.3
Aebi, U.4
Herrmann, H.5
-
41
-
-
31044432248
-
Telomere tethering at the nuclear periphery is essential for efficient DNA double strand break repair in subtelomeric region
-
Therizols P., Fairhead C., Cabal G.G., Genovesio A., Olivo-Marin J.C., Dujon B., et al. Telomere tethering at the nuclear periphery is essential for efficient DNA double strand break repair in subtelomeric region. J Cell Biol 172 (2006) 189-199
-
(2006)
J Cell Biol
, vol.172
, pp. 189-199
-
-
Therizols, P.1
Fairhead, C.2
Cabal, G.G.3
Genovesio, A.4
Olivo-Marin, J.C.5
Dujon, B.6
-
42
-
-
24644459728
-
Blocking protein farnesyltransferase improves nuclear shape in fibroblasts from humans with progeroid syndromes
-
Toth J.I., Yang S.H., Qiao X., Beigneux A.P., Gelb M.H., Moulson C.L., et al. Blocking protein farnesyltransferase improves nuclear shape in fibroblasts from humans with progeroid syndromes. Proc Nat Acad Sci USA 102 (2005) 12873-12878
-
(2005)
Proc Nat Acad Sci USA
, vol.102
, pp. 12873-12878
-
-
Toth, J.I.1
Yang, S.H.2
Qiao, X.3
Beigneux, A.P.4
Gelb, M.H.5
Moulson, C.L.6
-
43
-
-
25644440744
-
Accelerated ageing in mice deficient in Zmpste24 protease is linked to p53 signaling activation
-
Varela I., Cadinanos J., Pendas A.M., Gutierrez-Fernandez A., Folgueras A.R., Sanchez L.M., et al. Accelerated ageing in mice deficient in Zmpste24 protease is linked to p53 signaling activation. Nature 437 (2005) 564-568
-
(2005)
Nature
, vol.437
, pp. 564-568
-
-
Varela, I.1
Cadinanos, J.2
Pendas, A.M.3
Gutierrez-Fernandez, A.4
Folgueras, A.R.5
Sanchez, L.M.6
-
44
-
-
22544440839
-
Blocking protein farnesyltransferase improves nuclear blebbing in mouse fibroblasts with a targeted Hutchinson-Gilford progeria syndrome mutation
-
Yang S.H., Bergo M.O., Toth J.I., Qiao X., Hu Y., Sandoval S., et al. Blocking protein farnesyltransferase improves nuclear blebbing in mouse fibroblasts with a targeted Hutchinson-Gilford progeria syndrome mutation. Proc Nat Acad Sci USA 102 (2005) 10291-10296
-
(2005)
Proc Nat Acad Sci USA
, vol.102
, pp. 10291-10296
-
-
Yang, S.H.1
Bergo, M.O.2
Toth, J.I.3
Qiao, X.4
Hu, Y.5
Sandoval, S.6
|