-
1
-
-
20544436649
-
Sudden death in patients with myocardial infarction and left ventricular dysfunction, heart failure, or both
-
Solomon S.D., Zelenkofske S., McMurray J.J., Finn P.V., Velazquez E., Ertl G., Harsanyi A., Rouleau J.L., Maggioni A., Kober L., White H., Van de W.F., Pieper K., Califf R.M., and Pfeffer M.A. Sudden death in patients with myocardial infarction and left ventricular dysfunction, heart failure, or both. N Engl J Med 352 (2005) 2581-2588
-
(2005)
N Engl J Med
, vol.352
, pp. 2581-2588
-
-
Solomon, S.D.1
Zelenkofske, S.2
McMurray, J.J.3
Finn, P.V.4
Velazquez, E.5
Ertl, G.6
Harsanyi, A.7
Rouleau, J.L.8
Maggioni, A.9
Kober, L.10
White, H.11
Van de, W.F.12
Pieper, K.13
Califf, R.M.14
Pfeffer, M.A.15
-
2
-
-
33748640974
-
Familial sudden death is an important risk factor for primary ventricular fibrillation: a case-control study in acute myocardial infarction patients
-
Dekker L.R., Bezzina C.R., Henriques J.P., Tanck M.W., Koch K.T., Alings M.W., Arnold A.E., de Boer M.J., Gorgels A.P., Michels H.R., Verkerk A., Verheugt F.W., Zijlstra F., and Wilde A.A. Familial sudden death is an important risk factor for primary ventricular fibrillation: a case-control study in acute myocardial infarction patients. Circulation 114 (2006) 1140-1145
-
(2006)
Circulation
, vol.114
, pp. 1140-1145
-
-
Dekker, L.R.1
Bezzina, C.R.2
Henriques, J.P.3
Tanck, M.W.4
Koch, K.T.5
Alings, M.W.6
Arnold, A.E.7
de Boer, M.J.8
Gorgels, A.P.9
Michels, H.R.10
Verkerk, A.11
Verheugt, F.W.12
Zijlstra, F.13
Wilde, A.A.14
-
3
-
-
0026490286
-
Cellular and molecular biology of voltage-gated sodium channels
-
Catterall W.A. Cellular and molecular biology of voltage-gated sodium channels. Physiol Rev 72 (1992) S15-S48
-
(1992)
Physiol Rev
, vol.72
-
-
Catterall, W.A.1
-
4
-
-
2342523271
-
Human genomics and its impact on arrhythmias
-
Roden D.M. Human genomics and its impact on arrhythmias. Trends Cardiovasc Med 14 (2004) 112-116
-
(2004)
Trends Cardiovasc Med
, vol.14
, pp. 112-116
-
-
Roden, D.M.1
-
5
-
-
14644439241
-
Molecular genetics of arrhythmias and cardiovascular conditions associated with arrhythmias
-
Antzelevitch C. Molecular genetics of arrhythmias and cardiovascular conditions associated with arrhythmias. Heart Rhythm 1 (2004) 42C-56C
-
(2004)
Heart Rhythm
, vol.1
-
-
Antzelevitch, C.1
-
6
-
-
0032741905
-
Ionic mechanisms responsible for the electrocardiographic phenotype of the Brugada syndrome are temperature dependent
-
Dumaine R., Towbin J.A., Brugada P., Vatta M., Nesterenko D.V., Nesterenko V.V., Brugada J., Brugada R., and Antzelevitch C. Ionic mechanisms responsible for the electrocardiographic phenotype of the Brugada syndrome are temperature dependent. Circ Res 85 (1999) 803-809
-
(1999)
Circ Res
, vol.85
, pp. 803-809
-
-
Dumaine, R.1
Towbin, J.A.2
Brugada, P.3
Vatta, M.4
Nesterenko, D.V.5
Nesterenko, V.V.6
Brugada, J.7
Brugada, R.8
Antzelevitch, C.9
-
7
-
-
0034466612
-
Use of intravenous antiarrhythmics to identify concealed Brugada syndrome
-
Brugada R. Use of intravenous antiarrhythmics to identify concealed Brugada syndrome. Curr Control Trials Cardiovasc Med 1 (2000) 45-47
-
(2000)
Curr Control Trials Cardiovasc Med
, vol.1
, pp. 45-47
-
-
Brugada, R.1
-
8
-
-
32144452025
-
Provocation of sudden heart rate oscillation with adenosine exposes abnormal QT responses in patients with long QT syndrome: a bedside test for diagnosing long QT syndrome
-
Viskin S., Rosso R., Rogowski O., Belhassen B., Levitas A., Wagshal A., Katz A., Fourey D., Zeltser D., Oliva A., Pollevick G.D., Antzelevitch C., and Rozovski U. Provocation of sudden heart rate oscillation with adenosine exposes abnormal QT responses in patients with long QT syndrome: a bedside test for diagnosing long QT syndrome. Eur Heart J 27 (2006) 469-475
-
(2006)
Eur Heart J
, vol.27
, pp. 469-475
-
-
Viskin, S.1
Rosso, R.2
Rogowski, O.3
Belhassen, B.4
Levitas, A.5
Wagshal, A.6
Katz, A.7
Fourey, D.8
Zeltser, D.9
Oliva, A.10
Pollevick, G.D.11
Antzelevitch, C.12
Rozovski, U.13
-
9
-
-
22544461665
-
Susceptibility genes and modifiers for cardiac arrhythmias
-
Kaab S., and Schulze-Bahr E. Susceptibility genes and modifiers for cardiac arrhythmias. Cardiovasc Res 67 (2005) 397-413
-
(2005)
Cardiovasc Res
, vol.67
, pp. 397-413
-
-
Kaab, S.1
Schulze-Bahr, E.2
-
10
-
-
0345690174
-
Ethnic differences in cardiac potassium channel variants: implications for genetic susceptibility to sudden cardiac death and genetic testing for congenital long QT syndrome
-
Ackerman M.J., Tester D.J., Jones G.S., Will M.L., Burrow C.R., and Curran M.E. Ethnic differences in cardiac potassium channel variants: implications for genetic susceptibility to sudden cardiac death and genetic testing for congenital long QT syndrome. Mayo Clin Proc 78 (2003) 1479-1487
-
(2003)
Mayo Clin Proc
, vol.78
, pp. 1479-1487
-
-
Ackerman, M.J.1
Tester, D.J.2
Jones, G.S.3
Will, M.L.4
Burrow, C.R.5
Curran, M.E.6
-
11
-
-
0027425976
-
Differences in the electrophysiological response of canine ventricular epicardium and endocardium to ischemia: role of the transient outward current
-
Lukas A., and Antzelevitch C. Differences in the electrophysiological response of canine ventricular epicardium and endocardium to ischemia: role of the transient outward current. Circulation 88 (1993) 2903-2915
-
(1993)
Circulation
, vol.88
, pp. 2903-2915
-
-
Lukas, A.1
Antzelevitch, C.2
-
12
-
-
0030245546
-
Phase 2 reentry as a mechanism of initiation of circus movement reentry in canine epicardium exposed to simulated ischemia
-
Lukas A., and Antzelevitch C. Phase 2 reentry as a mechanism of initiation of circus movement reentry in canine epicardium exposed to simulated ischemia. Cardiovasc Res 32 (1996) 593-603
-
(1996)
Cardiovasc Res
, vol.32
, pp. 593-603
-
-
Lukas, A.1
Antzelevitch, C.2
-
13
-
-
0032879716
-
Cellular basis for the Brugada Syndrome and other mechanisms of arrhythmogenesis associated with ST segment elevation
-
Yan G.X., and Antzelevitch C. Cellular basis for the Brugada Syndrome and other mechanisms of arrhythmogenesis associated with ST segment elevation. Circulation 100 (1999) 1660-1666
-
(1999)
Circulation
, vol.100
, pp. 1660-1666
-
-
Yan, G.X.1
Antzelevitch, C.2
-
14
-
-
4844226196
-
Phase 2 reentry as a trigger to initiate ventricular fibrillation during early acute myocardial ischemia
-
Yan G.X., Joshi A., Guo D., Hlaing T., Martin J., Xu X., and Kowey P.R. Phase 2 reentry as a trigger to initiate ventricular fibrillation during early acute myocardial ischemia. Circulation 110 (2004) 1036-1041
-
(2004)
Circulation
, vol.110
, pp. 1036-1041
-
-
Yan, G.X.1
Joshi, A.2
Guo, D.3
Hlaing, T.4
Martin, J.5
Xu, X.6
Kowey, P.R.7
-
15
-
-
0038184640
-
Clinical Approaches to Tachyarrhythmias
-
Blackwell Futura Publishing Co, Armonk, NY
-
Antzelevitch C., Brugada P., Brugada J., Brugada R., Nademanee K., and Towbin J.A. Clinical Approaches to Tachyarrhythmias. The Brugada Syndrome (1999), Blackwell Futura Publishing Co, Armonk, NY 1-99
-
(1999)
The Brugada Syndrome
, pp. 1-99
-
-
Antzelevitch, C.1
Brugada, P.2
Brugada, J.3
Brugada, R.4
Nademanee, K.5
Towbin, J.A.6
-
16
-
-
0037145916
-
ST-segment elevation and ventricular fibrillation without coronary spasm by intracoronary injection of acetylcholine and/or ergonovine maleate in patients with Brugada syndrome
-
Noda T., Shimizu W., Taguchi A., Satomi K., Suyama K., Kurita T., Aihara N., and Kamakura S. ST-segment elevation and ventricular fibrillation without coronary spasm by intracoronary injection of acetylcholine and/or ergonovine maleate in patients with Brugada syndrome. J Am Coll Cardiol 40 (2002) 1841-1847
-
(2002)
J Am Coll Cardiol
, vol.40
, pp. 1841-1847
-
-
Noda, T.1
Shimizu, W.2
Taguchi, A.3
Satomi, K.4
Suyama, K.5
Kurita, T.6
Aihara, N.7
Kamakura, S.8
-
17
-
-
0032546384
-
Genetic basis and molecular mechanisms for idiopathic ventricular fibrillation
-
Chen Q., Kirsch G.E., Zhang D., Brugada R., Brugada J., Brugada P., Potenza D., Moya A., Borggrefe M., Breithardt G., Ortiz-Lopez R., Wang Z., Antzelevitch C., O'Brien R.E., Schultze-Bahr E., Keating M.T., Towbin J.A., and Wang Q. Genetic basis and molecular mechanisms for idiopathic ventricular fibrillation. Nature 392 (1998) 293-296
-
(1998)
Nature
, vol.392
, pp. 293-296
-
-
Chen, Q.1
Kirsch, G.E.2
Zhang, D.3
Brugada, R.4
Brugada, J.5
Brugada, P.6
Potenza, D.7
Moya, A.8
Borggrefe, M.9
Breithardt, G.10
Ortiz-Lopez, R.11
Wang, Z.12
Antzelevitch, C.13
O'Brien, R.E.14
Schultze-Bahr, E.15
Keating, M.T.16
Towbin, J.A.17
Wang, Q.18
-
18
-
-
1442356568
-
Sodium channel gene (SCN5A) mutations in 44 index patients with Brugada syndrome: different incidences in familial and sporadic disease
-
Schulze-Bahr E., Eckardt L., Breithardt G., Seidl K., Wichter T., Wolpert C., Borggrefe M., and Haverkamp W. Sodium channel gene (SCN5A) mutations in 44 index patients with Brugada syndrome: different incidences in familial and sporadic disease. Hum Mutat 21 (2003) 651-652
-
(2003)
Hum Mutat
, vol.21
, pp. 651-652
-
-
Schulze-Bahr, E.1
Eckardt, L.2
Breithardt, G.3
Seidl, K.4
Wichter, T.5
Wolpert, C.6
Borggrefe, M.7
Haverkamp, W.8
-
19
-
-
0037125369
-
Genotype-phenotype relationship in Brugada syndrome: electrocardiographic features differentiate SCN5A-related patients from non-SCN5A-related patients
-
Smits J.P., Eckardt L., Probst V., Bezzina C.R., Schott J.J., Remme C.A., Haverkamp W., Breithardt G., Escande D., Schulze-Bahr E., LeMarec H., and Wilde A.A. Genotype-phenotype relationship in Brugada syndrome: electrocardiographic features differentiate SCN5A-related patients from non-SCN5A-related patients. J Am Coll Cardiol 40 (2002) 350-356
-
(2002)
J Am Coll Cardiol
, vol.40
, pp. 350-356
-
-
Smits, J.P.1
Eckardt, L.2
Probst, V.3
Bezzina, C.R.4
Schott, J.J.5
Remme, C.A.6
Haverkamp, W.7
Breithardt, G.8
Escande, D.9
Schulze-Bahr, E.10
LeMarec, H.11
Wilde, A.A.12
-
20
-
-
23644450590
-
Genetics of acquired long QT syndrome
-
Roden D.M., and Viswanathan P.C. Genetics of acquired long QT syndrome. J Clin Invest 115 (2005) 2025-2032
-
(2005)
J Clin Invest
, vol.115
, pp. 2025-2032
-
-
Roden, D.M.1
Viswanathan, P.C.2
-
21
-
-
0037421629
-
A common human SCN5A polymorphism modifies expression of an arrhythmia causing mutation
-
Ye B., Valdivia C.R., Ackerman M.J., and Makielski J.C. A common human SCN5A polymorphism modifies expression of an arrhythmia causing mutation. Physiol Genomics 12 (2003) 187-193
-
(2003)
Physiol Genomics
, vol.12
, pp. 187-193
-
-
Ye, B.1
Valdivia, C.R.2
Ackerman, M.J.3
Makielski, J.C.4
-
22
-
-
0037314358
-
A common SCN5A polymorphism modulates the biophysical effects of an SCN5A mutation
-
Viswanathan P.C., Benson D.W., and Balser J.R. A common SCN5A polymorphism modulates the biophysical effects of an SCN5A mutation. J Clin Invest 111 (2003) 341-346
-
(2003)
J Clin Invest
, vol.111
, pp. 341-346
-
-
Viswanathan, P.C.1
Benson, D.W.2
Balser, J.R.3
-
23
-
-
33747146463
-
SCN5A Polymorphism restores trafficking of a Brugada syndrome mutation on a separate gene
-
Poelzing S., Forleo C., Samodell M., Dudash L., Sorrentino S., Anaclerio M., Troccoli R., Iacoviello M., Romito R., Guida P., Chahine M., Pitzalis M., and Deschenes I. SCN5A Polymorphism restores trafficking of a Brugada syndrome mutation on a separate gene. Circulation 114 (2006) 368-376
-
(2006)
Circulation
, vol.114
, pp. 368-376
-
-
Poelzing, S.1
Forleo, C.2
Samodell, M.3
Dudash, L.4
Sorrentino, S.5
Anaclerio, M.6
Troccoli, R.7
Iacoviello, M.8
Romito, R.9
Guida, P.10
Chahine, M.11
Pitzalis, M.12
Deschenes, I.13
-
24
-
-
0242330187
-
A ubiquitous splice variant and a common polymorphism affect heterologous expression of recombinant human SCN5A heart sodium channels
-
Makielski J.C., Ye B., Valdivia C.R., Pagel M.D., Pu J., Tester D.J., and Ackerman M.J. A ubiquitous splice variant and a common polymorphism affect heterologous expression of recombinant human SCN5A heart sodium channels. Circ Res 93 (2003) 821-828
-
(2003)
Circ Res
, vol.93
, pp. 821-828
-
-
Makielski, J.C.1
Ye, B.2
Valdivia, C.R.3
Pagel, M.D.4
Pu, J.5
Tester, D.J.6
Ackerman, M.J.7
-
25
-
-
21344433631
-
Common human SCN5A polymorphisms have altered electrophysiology when expressed in Q1077 splice variants
-
Tan B.H., Valdivia C.R., Rok B.A., Ye B., Ruwaldt K.M., Tester D.J., Ackerman M.J., and Makielski J.C. Common human SCN5A polymorphisms have altered electrophysiology when expressed in Q1077 splice variants. Heart Rhythm 2 (2005) 741-747
-
(2005)
Heart Rhythm
, vol.2
, pp. 741-747
-
-
Tan, B.H.1
Valdivia, C.R.2
Rok, B.A.3
Ye, B.4
Ruwaldt, K.M.5
Tester, D.J.6
Ackerman, M.J.7
Makielski, J.C.8
-
26
-
-
0019212141
-
Different electrophysiological responses of canine endocardium and epicardium to combined hyperkalemia, hypoxia, and acidosis
-
Gilmour Jr. R.F., and Zipes D.P. Different electrophysiological responses of canine endocardium and epicardium to combined hyperkalemia, hypoxia, and acidosis. Circ Res 46 (1980) 814-825
-
(1980)
Circ Res
, vol.46
, pp. 814-825
-
-
Gilmour Jr., R.F.1
Zipes, D.P.2
-
27
-
-
0022459840
-
Simultaneous recording of action potentials from endocardium and epicardium during ischemia in the isolated cat ventricle: relation of temporal electrophysiologic heterogeneities to arrhythmias
-
Kimura S., Bassett A.L., Kohya T., Kozlovskis P.L., and Myerburg R.J. Simultaneous recording of action potentials from endocardium and epicardium during ischemia in the isolated cat ventricle: relation of temporal electrophysiologic heterogeneities to arrhythmias. Circulation 74 (1986) 401-409
-
(1986)
Circulation
, vol.74
, pp. 401-409
-
-
Kimura, S.1
Bassett, A.L.2
Kohya, T.3
Kozlovskis, P.L.4
Myerburg, R.J.5
-
28
-
-
0027320016
-
Pinacidil-induced electrical heterogeneity and extrasystolic activity in canine ventricular tissues. Does activation of ATP-regulated potassium current promote phase 2 reentry?
-
Di Diego J.M., and Antzelevitch C. Pinacidil-induced electrical heterogeneity and extrasystolic activity in canine ventricular tissues. Does activation of ATP-regulated potassium current promote phase 2 reentry?. Circulation 88 (1993) 1177-1189
-
(1993)
Circulation
, vol.88
, pp. 1177-1189
-
-
Di Diego, J.M.1
Antzelevitch, C.2
-
29
-
-
0028182422
-
High [Ca2+]-induced electrical heterogeneity and extrasystolic activity in isolated canine ventricular epicardium. Phase 2 reentry
-
Di Diego J.M., and Antzelevitch C. High [Ca2+]-induced electrical heterogeneity and extrasystolic activity in isolated canine ventricular epicardium. Phase 2 reentry. Circulation 89 (1994) 1839-1850
-
(1994)
Circulation
, vol.89
, pp. 1839-1850
-
-
Di Diego, J.M.1
Antzelevitch, C.2
-
30
-
-
0027092442
-
Ro 40-5967, a novel calcium channel antagonist, protects against ventricular fibrillation
-
Billman G.E. Ro 40-5967, a novel calcium channel antagonist, protects against ventricular fibrillation. Eur J Pharmacol 229 (1992) 179-187
-
(1992)
Eur J Pharmacol
, vol.229
, pp. 179-187
-
-
Billman, G.E.1
-
31
-
-
0027460628
-
Ionic bases for electrophysiological distinctions among epicardial, midmyocardial, and endocardial myocytes from the free wall of the canine left ventricle
-
Liu D.W., Gintant G.A., and Antzelevitch C. Ionic bases for electrophysiological distinctions among epicardial, midmyocardial, and endocardial myocytes from the free wall of the canine left ventricle. Circ Res 72 (1993) 671-687
-
(1993)
Circ Res
, vol.72
, pp. 671-687
-
-
Liu, D.W.1
Gintant, G.A.2
Antzelevitch, C.3
-
32
-
-
0029756180
-
to and action potential notch are smaller in left vs. right canine ventricular epicardium
-
to and action potential notch are smaller in left vs. right canine ventricular epicardium. Am J Physiol 271 (1996) H548-H561
-
(1996)
Am J Physiol
, vol.271
-
-
Di Diego, J.M.1
Sun, Z.Q.2
Antzelevitch, C.3
-
33
-
-
0347989484
-
Cellular basis for ST-segment changes observed during ischemia
-
Di Diego J.M., and Antzelevitch C. Cellular basis for ST-segment changes observed during ischemia. J Electrocardiol 36 Suppl (2003) 1-5
-
(2003)
J Electrocardiol
, vol.36
, Issue.SUPPL
, pp. 1-5
-
-
Di Diego, J.M.1
Antzelevitch, C.2
|