The role of CSA in the response to oxidative DNA damage in human cells

Oncogene

Volumn 26, Issue 30, 2007, Pages 4336-4343

  D'Errico, M ^a   Parlanti, E ^a   Teson, M ^b   Degan, P ^c   Lemma, T ^a   Calcagnile, A ^a   Iavarone, I ^a   Jaruga, P ^d,e   Ropolo, M ^c   Pedrini, A M ^f   Orioli, D ^f   Frosina, G ^c   Zambruno, G ^b   Dizdaroglu, M ^e   Stefanini, M ^f   Dogliotti, E ^a  

^a ISTITUTO SUPERIORE DI SANITÀ   (Italy)

^b LABORATORIO DI PATOLOGIA VASCOLARE   (Italy)

^c UNIVERSITY OF GENOA   (Italy)

^d University of Marylandw Baltimore County   (United States)

^e NATIONAL INSTITUTE OF STANDARDS AND TECHNOLOGY   (United States)

^f CNR   (Italy)

Author keywords

Cockayne syndrome; DNA repair; Keratinocytes; Oxidative DNA damage

Indexed keywords

8 HYDROXYGUANINE; CELL EXTRACT; COCKAYNE SYNDROME A PROTEIN; COCKAYNE SYNDROME B PROTEIN; DEOXYADENOSINE DERIVATIVE; POTASSIUM BROMATE; PROTEIN; UNCLASSIFIED DRUG;

ARTICLE; CONTROLLED STUDY; DNA DAMAGE; FIBROBLAST; GENE FUNCTION; HUMAN; HUMAN CELL; KERATINOCYTE; OXIDATIVE STRESS; PRIORITY JOURNAL; STEADY STATE; WILD TYPE;

DEOXYGUANOSINE; DNA DAMAGE; DNA HELICASES; DNA REPAIR; DNA REPAIR ENZYMES; FIBROBLASTS; HUMANS; KERATINOCYTES; OXIDATION-REDUCTION; TRANSCRIPTION FACTORS;

EID: 34347352111     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/sj.onc.1210232     Document Type: Article

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