Macrophages and neutrophils are the targets for immune suppression by glucocorticoids in contact allergy

Journal of Clinical Investigation

Volumn 117, Issue 5, 2007, Pages 1381-1390

  Tuckermann, Jan P ^a,b   Kleiman, Anna ^a,b   Moriggl, Richard ^c   Spanbroek, Rainer ^d   Neumann, Anita ^b   Illing, Anett ^b   Clausen, Björn E ^e   Stride, Brenda ^a,f   Förster, Irmgard ^g   Habenicht, Andreas J R ^d   Reichardt, Holger M ^h   Tronche, François ⁱ   Schmid, Wolfgang ^a   Schütz, Günther ^a  

^a GERMAN CANCER RESEARCH CENTER DKFZ   (Germany)

^b FRITZ LIPMANN INSTITUTE   (Germany)

^c MEDICAL UNIVERSITY OF VIENNA   (Austria)

^d FRIEDRICH SCHILLER UNIVERSITY JENA   (Germany)

^e UNIVERSITY OF AMSTERDAM   (Netherlands)

^f EUROPEAN MOLECULAR BIOLOGY LABORATORY   (Germany)

^g HEINRICH HEINE UNIVERSITY   (Germany)

^h UNIVERSITY OF GÖTTINGEN   (Germany)

ⁱ CNRS   (France)

Author keywords

[No Author keywords available]

Indexed keywords

1 FLUORO 2,4 DINITROBENZENE; CRE RECOMBINASE; DEXAMETHASONE; FLUOCINONIDE; GAMMA INTERFERON INDUCIBLE PROTEIN 10; GLUCOCORTICOID; GLUCOCORTICOID RECEPTOR; HAPTEN; INTERLEUKIN 1BETA; MACROPHAGE INFLAMMATORY PROTEIN 2; MONOCYTE CHEMOTACTIC PROTEIN 1; OXAZOLONE; RECOMBINANT TUMOR NECROSIS FACTOR ALPHA; TAMOXIFEN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIINFLAMMATORY ACTIVITY; ARTICLE; BONE MARROW CELL; CELL TYPE; CONTACT ALLERGY; CONTROLLED STUDY; DIMERIZATION; DNA BINDING; DOWN REGULATION; DRUG RESISTANCE; DRUG TARGETING; GENE EXPRESSION; GENE INACTIVATION; IMMUNOSUPPRESSIVE TREATMENT; INFLAMMATION; KERATINOCYTE; LYMPHOCYTIC INFILTRATION; MACROPHAGE; MACROPHAGE MIGRATION; MOLECULAR MECHANICS; MOUSE; NEUTROPHIL; NEUTROPHIL CHEMOTAXIS; NONHUMAN; PRIORITY JOURNAL; T LYMPHOCYTE; TISSUE ENGINEERING;

ANIMALS; ANTI-INFLAMMATORY AGENTS; CELLS, CULTURED; DERMATITIS, ALLERGIC CONTACT; DEXAMETHASONE; DRUG DELIVERY SYSTEMS; IMMUNOSUPPRESSIVE AGENTS; MACROPHAGES; MICE; MICE, INBRED BALB C; MICE, INBRED C57BL; MICE, TRANSGENIC; NEUTROPHILS; RECEPTORS, GLUCOCORTICOID;

EID: 34248206597     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI28034     Document Type: Article

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