Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis [1]

Cell Death and Differentiation

Volumn 14, Issue 5, 2007, Pages 1060-1062

  Ceci, M ^a   Gallo, P ^b,c   Santonastasi, M ^a,c   Grimaldi, S ^a,c   Latronico, M V G ^a   Pitisci, A ^a,c   Missol Kolka, E ^d   Scimia, M C ^a,e   Catalucci, D ^a,e   Hilfiker Kleiner, D ^d   Condorelli, G ^a,e  

^a IRCCS MULTIMEDICA   (Italy)

^b UNIVERSITY CAMPUS BIO MEDICO   (Italy)

^c S Raffaele Biomedical Park Foundation   (Italy)

^d HANNOVER MEDICAL SCHOOL   (Germany)

^e UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN E40K; PROTEIN KINASE B; UNCLASSIFIED DRUG; VASCULOTROPIN;

ANGIOGENESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; AORTA CONSTRICTION; APOPTOSIS; CAPILLARY DENSITY; CONTROLLED STUDY; GENE OVEREXPRESSION; HEART FAILURE; HEART FUNCTION; HEART MUSCLE; HISTOLOGY; LETTER; MOUSE; MUTANT; NONHUMAN; PRIORITY JOURNAL; VASCULARIZATION; WESTERN BLOTTING;

ANIMALS; APOPTOSIS; BLOOD PRESSURE; CARDIAC OUTPUT, LOW; CORONARY CIRCULATION; GENE EXPRESSION REGULATION; GLUTAMIC ACID; LYSINE; MICE; MYOCARDIUM; MYOCYTES, CARDIAC; NEOVASCULARIZATION, PHYSIOLOGIC; PROTO-ONCOGENE PROTEINS C-AKT; RNA, MESSENGER; VASCULAR ENDOTHELIAL GROWTH FACTOR A;

MUS;

EID: 34247395400     PISSN: 13509047     EISSN: 14765403     Source Type: Journal    
DOI: 10.1038/sj.cdd.4402095     Document Type: Letter

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