Copper ions disrupt dopamine metabolism via inhibition of V-H +-ATPase: A possible contributing factor to neurotoxicity

Journal of Neurochemistry

Volumn 101, Issue 2, 2007, Pages 313-326

  Wimalasena, D Shyamali ^a   Wiese, Thomas J ^b   Wimalasena, Kandatege ^a  

^a WICHITA STATE UNIVERSITY   (United States)

^b FORT HAYS STATE UNIVERSITY   (United States)

Author keywords

Catecholamines; Chromaffin granules; Copper; Neurodegenerative diseases; SH SY5Y cells.; Vesicular H+ ATPase

Indexed keywords

AMYLOID BETA PROTEIN; ASCORBIC ACID; CATECHOLAMINE; CHELATING AGENT; COPPER ION; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATASE; REACTIVE OXYGEN METABOLITE;

ANIMAL CELL; ARTICLE; CHROMAFFIN GRANULE; CONTROLLED STUDY; DOPAMINE METABOLISM; ENZYME INHIBITION; HUMAN; HUMAN CELL; NEUROTOXICITY; NONHUMAN; PATHOPHYSIOLOGY; PH MEASUREMENT; PRIORITY JOURNAL;

ANIMALS; BRAIN; CATTLE; CELL LINE, TUMOR; CHROMAFFIN GRANULES; COPPER; DOPAMINE; ENZYME INHIBITORS; HUMANS; HYDROGEN-ION CONCENTRATION; NEURODEGENERATIVE DISEASES; NEURONS; NEUROTOXINS; PROTON-TRANSLOCATING ATPASES; SUBCELLULAR FRACTIONS;

EID: 34047178993     PISSN: 00223042     EISSN: 14714159     Source Type: Journal    
DOI: 10.1111/j.1471-4159.2006.04362.x     Document Type: Article

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