-
1
-
-
0034614897
-
Consequences of cell death: Exposure to necrotic tumor cells, but not primary tissue cells or apoptotic cells, induces the maturation of immunostimulatory dendritic cells
-
Sauter B, Albert ML, Francisco L et al (2000) Consequences of cell death: exposure to necrotic tumor cells, but not primary tissue cells or apoptotic cells, induces the maturation of immunostimulatory dendritic cells. J Exp Med 191:423-434
-
(2000)
J Exp Med
, vol.191
, pp. 423-434
-
-
Sauter, B.1
Albert, M.L.2
Francisco, L.3
-
2
-
-
29144530630
-
Caspase-dependent immunogenicity of doxorubicin-induced tumor cell death
-
Casares N, Pequignot MO, Tesniere A et al (2005) Caspase-dependent immunogenicity of doxorubicin-induced tumor cell death. J Exp Med 202:1691-1701
-
(2005)
J Exp Med
, vol.202
, pp. 1691-1701
-
-
Casares, N.1
Pequignot, M.O.2
Tesniere, A.3
-
4
-
-
33846057130
-
Calreticulin exposure dictates the immunogenicity of cancer cell death
-
Obeid M, Tesniere A, Ghiringhelli F et al (2007) Calreticulin exposure dictates the immunogenicity of cancer cell death. Nat Med 13:54-61
-
(2007)
Nat Med
, vol.13
, pp. 54-61
-
-
Obeid, M.1
Tesniere, A.2
Ghiringhelli, F.3
-
5
-
-
33344476071
-
Regulation of peripheral T cell activation by calreticulin
-
Porcellini S, Traggiai F, Schenk U et al (2006) Regulation of peripheral T cell activation by calreticulin. J Exp Med 203:461-471
-
(2006)
J Exp Med
, vol.203
, pp. 461-471
-
-
Porcellini, S.1
Traggiai, F.2
Schenk, U.3
-
6
-
-
0027507325
-
The calcium-binding protein calreticulin is a major constituent of lytic granules in cytolytic T lymphocytes
-
Dupuis M, Schaerer E, Krause KH, Tschopp J (1993) The calcium-binding protein calreticulin is a major constituent of lytic granules in cytolytic T lymphocytes. J Exp Med 177:1-7
-
(1993)
J Exp Med
, vol.177
, pp. 1-7
-
-
Dupuis, M.1
Schaerer, E.2
Krause, K.H.3
Tschopp, J.4
-
7
-
-
0032555064
-
2+-binding protein calreticulin and perforin, a component of the cytotoxic T-cell granules
-
2+-binding protein calreticulin and perforin, a component of the cytotoxic T-cell granules. Biochemistry 37:10386-10394
-
(1998)
Biochemistry
, vol.37
, pp. 10386-10394
-
-
Andrin, C.1
Pinkoski, M.J.2
Burns, K.3
-
8
-
-
14844352487
-
Impaired cytolytic activity in calreticulin-deficient CTLs
-
Sipione S, Ewen C, Shostak I, Michalak M, Bleackley RC (2005) Impaired cytolytic activity in calreticulin-deficient CTLs. J Immunol 174:3212-3219
-
(2005)
J Immunol
, vol.174
, pp. 3212-3219
-
-
Sipione, S.1
Ewen, C.2
Shostak, I.3
Michalak, M.4
Bleackley, R.C.5
-
9
-
-
33644842388
-
Role of complement and other innate immune mechanisms in the removal of apoptotic cells
-
Ogden CA, Elkon KB (2006) Role of complement and other innate immune mechanisms in the removal of apoptotic cells. Curr Dir Autoimmun 9:120-142
-
(2006)
Curr Dir Autoimmun
, vol.9
, pp. 120-142
-
-
Ogden, C.A.1
Elkon, K.B.2
-
11
-
-
0032485487
-
Dendritic cells acquire antigen from apoptotic cells and induce class I-restricted CTLs
-
Albert ML, Sauter B, Bhardwaj N (1998) Dendritic cells acquire antigen from apoptotic cells and induce class I-restricted CTLs. Nature 392:86-89
-
(1998)
Nature
, vol.392
, pp. 86-89
-
-
Albert, M.L.1
Sauter, B.2
Bhardwaj, N.3
-
12
-
-
33750605140
-
Natural killer cells play a key role in the antitumor immunity generated by chaperone-rich cell lysate vaccination
-
Zeng Y, Chen X, Larmonier N et al (2006) Natural killer cells play a key role in the antitumor immunity generated by chaperone-rich cell lysate vaccination. Int J Can 119:2624-2631
-
(2006)
Int J Can
, vol.119
, pp. 2624-2631
-
-
Zeng, Y.1
Chen, X.2
Larmonier, N.3
-
13
-
-
0033747044
-
Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kappa B pathway
-
Basu S, Binder RJ, Suto R, Anderson KM, Srivastava PK (2000) Necrotic but not apoptotic cell death releases heat shock proteins, which deliver a partial maturation signal to dendritic cells and activate the NF-kappa B pathway. Int Immunol 12:1539-1546
-
(2000)
Int Immunol
, vol.12
, pp. 1539-1546
-
-
Basu, S.1
Binder, R.J.2
Suto, R.3
Anderson, K.M.4
Srivastava, P.K.5
-
14
-
-
0035070198
-
CD91 is a common receptor for heat shock proteins gp96, hsp90, hsp70, and calreticulin
-
Basu S, Binder RJ, Ramalingam T, Srivastava PK (2001) CD91 is a common receptor for heat shock proteins gp96, hsp90, hsp70, and calreticulin. Immunity 14:303-313
-
(2001)
Immunity
, vol.14
, pp. 303-313
-
-
Basu, S.1
Binder, R.J.2
Ramalingam, T.3
Srivastava, P.K.4
-
18
-
-
0032724653
-
Caspase-3 is the primary activator of apoptotic DNA fragmentation via DNA fragmentation factor-45/inhibitor of caspase-activated DNase inactivation
-
Wolf BB, Schuler M, Echeverri F, Green DR (1999) Caspase-3 is the primary activator of apoptotic DNA fragmentation via DNA fragmentation factor-45/inhibitor of caspase-activated DNase inactivation. J Biol Chem 274:30651-30656
-
(1999)
J Biol Chem
, vol.274
, pp. 30651-30656
-
-
Wolf, B.B.1
Schuler, M.2
Echeverri, F.3
Green, D.R.4
-
19
-
-
0035831473
-
Executioner caspase-3, -6, and -7 perform distinct, non-redundant roles during the demolition phase of apoptosis
-
Slee EA, Adrain C, Martin SJ (2001) Executioner caspase-3, -6, and -7 perform distinct, non-redundant roles during the demolition phase of apoptosis. J Biol Chem 276:7320-7326
-
(2001)
J Biol Chem
, vol.276
, pp. 7320-7326
-
-
Slee, E.A.1
Adrain, C.2
Martin, S.J.3
-
20
-
-
33747190755
-
Cell death provoked by loss of interleukin-3 signaling is independent of Bad, Bim, and PI3 kinase, but depends in part on Puma
-
Ekert PG, Jabbour AM, Manoharan A et al (2006) Cell death provoked by loss of interleukin-3 signaling is independent of Bad, Bim, and PI3 kinase, but depends in part on Puma. Blood 108:1461-1468
-
(2006)
Blood
, vol.108
, pp. 1461-1468
-
-
Ekert, P.G.1
Jabbour, A.M.2
Manoharan, A.3
-
21
-
-
0034630161
-
The CD95 (APO-1/Fas) and the TRAIL (APO-2L) apoptosis systems
-
Walczak H, Krammer PH (2000) The CD95 (APO-1/Fas) and the TRAIL (APO-2L) apoptosis systems. Exp Cell Res 256:58-66
-
(2000)
Exp Cell Res
, vol.256
, pp. 58-66
-
-
Walczak, H.1
Krammer, P.H.2
-
22
-
-
33645742877
-
Cytotoxic T lymphocyte-induced killing in the absence of granzymes A and B is unique and distinct from both apoptosis and perforin-dependent lysis
-
Waterhouse NJ, Sutton VR, Sedelies KA et al (2006) Cytotoxic T lymphocyte-induced killing in the absence of granzymes A and B is unique and distinct from both apoptosis and perforin-dependent lysis. J Cell Biol 173:133-144
-
(2006)
J Cell Biol
, vol.173
, pp. 133-144
-
-
Waterhouse, N.J.1
Sutton, V.R.2
Sedelies, K.A.3
-
23
-
-
0034630347
-
Heart, brain, and body wall defects in mice lacking calreticulin
-
Rauch F, Prud'homme J, Arabian A, Dedhar S, St-Arnaud R (2000) Heart, brain, and body wall defects in mice lacking calreticulin. Exp Cell Res 256:105-111
-
(2000)
Exp Cell Res
, vol.256
, pp. 105-111
-
-
Rauch, F.1
Prud'homme, J.2
Arabian, A.3
Dedhar, S.4
St-Arnaud, R.5
-
24
-
-
14544290056
-
Two pathways converge at CED-10 to mediate actin rearrangement and corpse removal in C. elegans
-
Kinchen JM, Cabello J, Klingele D et al (2005) Two pathways converge at CED-10 to mediate actin rearrangement and corpse removal in C. elegans. Nature 434:93-99
-
(2005)
Nature
, vol.434
, pp. 93-99
-
-
Kinchen, J.M.1
Cabello, J.2
Klingele, D.3
-
25
-
-
27744528518
-
Cancer cell immune escape and tumor progression by exploitation of anti-inflammatory and pro-inflammatory responses
-
Kim R, Emi M, Tanabe K (2005) Cancer cell immune escape and tumor progression by exploitation of anti-inflammatory and pro-inflammatory responses. Cancer Biol Ther 4:924-933
-
(2005)
Cancer Biol Ther
, vol.4
, pp. 924-933
-
-
Kim, R.1
Emi, M.2
Tanabe, K.3
|