-
1
-
-
0142056753
-
Structure, regulation, and function of mammalian membrane guanylyl cyclase receptors, with a focus on guanylyl cyclase-A
-
Kuhn M. Structure, regulation, and function of mammalian membrane guanylyl cyclase receptors, with a focus on guanylyl cyclase-A. Circ Res 93 (2003) 700-709
-
(2003)
Circ Res
, vol.93
, pp. 700-709
-
-
Kuhn, M.1
-
2
-
-
0038814317
-
Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A
-
Holtwick R., van Eickels M., Skryabin B.V., Baba H.A., Bubikat A., Begrow F., et al. Pressure-independent cardiac hypertrophy in mice with cardiomyocyte-restricted inactivation of the atrial natriuretic peptide receptor guanylyl cyclase-A. J Clin Invest 111 (2003) 1399-1407
-
(2003)
J Clin Invest
, vol.111
, pp. 1399-1407
-
-
Holtwick, R.1
van Eickels, M.2
Skryabin, B.V.3
Baba, H.A.4
Bubikat, A.5
Begrow, F.6
-
3
-
-
0035956914
-
A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy
-
Kishimoto I., Rossi K., and Garbers D.L. A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy. Proc Natl Acad Sci U S A 98 (2001) 2703-2706
-
(2001)
Proc Natl Acad Sci U S A
, vol.98
, pp. 2703-2706
-
-
Kishimoto, I.1
Rossi, K.2
Garbers, D.L.3
-
4
-
-
12944329901
-
Cardiac fibrosis in mice lacking brain natriuretic peptide
-
Tamura N., Ogawa Y., Chusho H., Nakamura K., Nakao K., Suda M., et al. Cardiac fibrosis in mice lacking brain natriuretic peptide. Proc Natl Acad Sci U S A 97 (2000) 4239-4244
-
(2000)
Proc Natl Acad Sci U S A
, vol.97
, pp. 4239-4244
-
-
Tamura, N.1
Ogawa, Y.2
Chusho, H.3
Nakamura, K.4
Nakao, K.5
Suda, M.6
-
5
-
-
5444222884
-
Left ventricular assist device support reverses altered cardiac expression and function of natriuretic peptides and receptors in end-stage heart failure
-
Kuhn M., Voss M., Mitko D., Stypmann J., Schmid C., Kawaguchi N., et al. Left ventricular assist device support reverses altered cardiac expression and function of natriuretic peptides and receptors in end-stage heart failure. Cardiovasc Res 64 (2004) 308-314
-
(2004)
Cardiovasc Res
, vol.64
, pp. 308-314
-
-
Kuhn, M.1
Voss, M.2
Mitko, D.3
Stypmann, J.4
Schmid, C.5
Kawaguchi, N.6
-
6
-
-
33747605830
-
Novel snake venom ligand dendroaspis natriuretic peptide is selective for natriuretic peptide receptor-A in human heart: downregulation of natriuretic peptide receptor-A in heart failure
-
Singh G., Kuc R.E., Maguire J.J., Fidock M., and Davenport A.P. Novel snake venom ligand dendroaspis natriuretic peptide is selective for natriuretic peptide receptor-A in human heart: downregulation of natriuretic peptide receptor-A in heart failure. Circ Res 99 (2006) 183-190
-
(2006)
Circ Res
, vol.99
, pp. 183-190
-
-
Singh, G.1
Kuc, R.E.2
Maguire, J.J.3
Fidock, M.4
Davenport, A.P.5
-
7
-
-
0028973389
-
Salt-resistant hypertension in mice lacking the guanylyl cyclase-A receptor for atrial natriuretic peptide
-
Lopez M.J., Wong S.K., Kishimoto I., Dubois S., Mach V., Friesen J., et al. Salt-resistant hypertension in mice lacking the guanylyl cyclase-A receptor for atrial natriuretic peptide. Nature 378 (1995) 65-68
-
(1995)
Nature
, vol.378
, pp. 65-68
-
-
Lopez, M.J.1
Wong, S.K.2
Kishimoto, I.3
Dubois, S.4
Mach, V.5
Friesen, J.6
-
8
-
-
13144306072
-
Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor A
-
Oliver P.M., Fox J.E., Kim R., Rockman H.A., Kim H.S., Reddick R.L., et al. Hypertension, cardiac hypertrophy, and sudden death in mice lacking natriuretic peptide receptor A. Proc Natl Acad Sci U S A 94 (1997) 14730-14735
-
(1997)
Proc Natl Acad Sci U S A
, vol.94
, pp. 14730-14735
-
-
Oliver, P.M.1
Fox, J.E.2
Kim, R.3
Rockman, H.A.4
Kim, H.S.5
Reddick, R.L.6
-
9
-
-
4444365354
-
Hypervolemic hypertension in mice with systemic inactivation of the (floxed) guanylyl cyclase-A gene by alphaMHC-Cre-mediated recombination
-
Skryabin B.V., Holtwick R., Fabritz L., Kruse M.N., Veltrup I., Stypmann J., et al. Hypervolemic hypertension in mice with systemic inactivation of the (floxed) guanylyl cyclase-A gene by alphaMHC-Cre-mediated recombination. Genesis 39 (2004) 288-298
-
(2004)
Genesis
, vol.39
, pp. 288-298
-
-
Skryabin, B.V.1
Holtwick, R.2
Fabritz, L.3
Kruse, M.N.4
Veltrup, I.5
Stypmann, J.6
-
10
-
-
0036213605
-
Progressive cardiac hypertrophy and dysfunction in atrial natriuretic peptide receptor (GC-A) deficient mice
-
Kuhn M., Holtwick R., Baba H.A., Perriard J.C., Schmitz W., and Ehler E. Progressive cardiac hypertrophy and dysfunction in atrial natriuretic peptide receptor (GC-A) deficient mice. Heart 87 (2002) 368-374
-
(2002)
Heart
, vol.87
, pp. 368-374
-
-
Kuhn, M.1
Holtwick, R.2
Baba, H.A.3
Perriard, J.C.4
Schmitz, W.5
Ehler, E.6
-
11
-
-
0035827719
-
Arrhythmogenesis and contractile dysfunction in heart failure: roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic responsiveness
-
Pogwizd S.M., Schlotthauer K., Li L., Yuan W., and Bers D.M. Arrhythmogenesis and contractile dysfunction in heart failure: roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic responsiveness. Circ Res 88 (2001) 1159-1167
-
(2001)
Circ Res
, vol.88
, pp. 1159-1167
-
-
Pogwizd, S.M.1
Schlotthauer, K.2
Li, L.3
Yuan, W.4
Bers, D.M.5
-
13
-
-
3042659100
-
Subtype-specific β-adrenoceptor signaling pathways in the heart and their potential clinical implications
-
Xiao R.P., Zhu W., Zheng M., Chakir K., Bond R., Lakatta E.G., et al. Subtype-specific β-adrenoceptor signaling pathways in the heart and their potential clinical implications. Trends Pharmacol Sci 25 (2004) 358-365
-
(2004)
Trends Pharmacol Sci
, vol.25
, pp. 358-365
-
-
Xiao, R.P.1
Zhu, W.2
Zheng, M.3
Chakir, K.4
Bond, R.5
Lakatta, E.G.6
-
14
-
-
33644865926
-
Increased sarcoplasmic reticulum calcium leak but unaltered contractility by acute CaMKII overexpression in isolated rabbit cardiac myocytes
-
Kohlhaas M., Zhang T., Seidler T., Zibrova D., Dybkova N., Steen A., et al. Increased sarcoplasmic reticulum calcium leak but unaltered contractility by acute CaMKII overexpression in isolated rabbit cardiac myocytes. Circ Res 98 (2006) 235-244
-
(2006)
Circ Res
, vol.98
, pp. 235-244
-
-
Kohlhaas, M.1
Zhang, T.2
Seidler, T.3
Zibrova, D.4
Dybkova, N.5
Steen, A.6
-
15
-
-
0034502240
-
The cardiac beta-adrenoceptor-G-protein(s)-adenylyl cyclase system in monocrotaline-treated rats
-
Seyfarth T., Gerbershagen H.P., Giessler C., Leineweber K., Heinroth-Hoffmann I., Ponicke K., et al. The cardiac beta-adrenoceptor-G-protein(s)-adenylyl cyclase system in monocrotaline-treated rats. J Mol Cell Cardiol 32 (2000) 2315-2326
-
(2000)
J Mol Cell Cardiol
, vol.32
, pp. 2315-2326
-
-
Seyfarth, T.1
Gerbershagen, H.P.2
Giessler, C.3
Leineweber, K.4
Heinroth-Hoffmann, I.5
Ponicke, K.6
-
16
-
-
0036167330
-
Dual site phospholamban phosphorylation and its physiological relevance in the heart
-
Hagemann D., and Xiao R.-P. Dual site phospholamban phosphorylation and its physiological relevance in the heart. Trends Cardiovasc Med 12 (2002) 51-56
-
(2002)
Trends Cardiovasc Med
, vol.12
, pp. 51-56
-
-
Hagemann, D.1
Xiao, R.-P.2
-
19
-
-
0034640113
-
PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts
-
Marx S.O., Reiken S., Hisamatsu Y., Jayaraman T., Burkhoff D., Rosemblit N., et al. PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts. Cell 101 (2000) 365-376
-
(2000)
Cell
, vol.101
, pp. 365-376
-
-
Marx, S.O.1
Reiken, S.2
Hisamatsu, Y.3
Jayaraman, T.4
Burkhoff, D.5
Rosemblit, N.6
-
21
-
-
33646771721
-
Ser-2030, but not Ser-2808, is the major phosphorylation site in cardiac ryanodine receptors responding to protein kinase A activation upon β-adrenergic stimulation in normal and failing hearts
-
Xiao B., Zhong G., Obayashi M., Yang D., Chen K., Walsh M.P., et al. Ser-2030, but not Ser-2808, is the major phosphorylation site in cardiac ryanodine receptors responding to protein kinase A activation upon β-adrenergic stimulation in normal and failing hearts. Biochem J 396 (2006) 7-16
-
(2006)
Biochem J
, vol.396
, pp. 7-16
-
-
Xiao, B.1
Zhong, G.2
Obayashi, M.3
Yang, D.4
Chen, K.5
Walsh, M.P.6
-
24
-
-
0033968324
-
The role of the cytoskeleton in heart failure
-
Hein S., Kostin S., and Heling A. The role of the cytoskeleton in heart failure. Cardiovasc Res 45 (2000) 273-278
-
(2000)
Cardiovasc Res
, vol.45
, pp. 273-278
-
-
Hein, S.1
Kostin, S.2
Heling, A.3
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