Hearts in adult offspring of copper-deficient dams exhibit decreased cytochrome c oxidase activity, increased mitochondrial hydrogen peroxide generation and enhanced formation of intracellular residual bodies

Journal of Nutritional Biochemistry

Volumn 18, Issue 2, 2007, Pages 97-104

  Johnson, W Thomas ^a   Newman Jr , Samuel M ^a  

^a GRAND FORKS HUMAN NUTRITION RESEARCH CENTER   (United States)

Author keywords

Copper deficiency; Cytochrome c oxidase; Heart; Mitochondria; Pregnancy; Rat pups

Indexed keywords

COPPER; CYTOCHROME C OXIDASE; HYDROGEN PEROXIDE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATOMIC ABSORPTION SPECTROMETRY; CELL SIZE; CELL VOLUME; CONTROLLED STUDY; COPPER DEFICIENCY; ENZYME ACTIVITY; FEMALE; HEART MITOCHONDRION; HEART MUSCLE CELL; LACTATION; MORPHOMETRICS; NONHUMAN; OXIDATIVE STRESS; PREGNANCY; PROGENY; RAT; STATISTICAL ANALYSIS; TRANSMISSION ELECTRON MICROSCOPY;

ANIMALS; COPPER; DIET; ELECTRON TRANSPORT COMPLEX IV; FEMALE; HYDROGEN PEROXIDE; MALE; MICROSCOPY, ELECTRON; MITOCHONDRIA, HEART; MYOCARDIUM; MYOCYTES, CARDIAC; PREGNANCY; PRENATAL EXPOSURE DELAYED EFFECTS; RATS; RATS, SPRAGUE-DAWLEY;

RATTUS;

EID: 33845966063     PISSN: 09552863     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.jnutbio.2006.03.005     Document Type: Article

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