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Volumn 47, Issue 12, 2006, Pages 2657-2658

JAK2 V617F mutation is absent in chronic lymphocytic leukemia [5]

Author keywords

[No Author keywords available]

Indexed keywords

GENOMIC DNA; JANUS KINASE 2; PHENYLALANINE; PROTEIN KINASE ZAP 70; STAT1 PROTEIN; STAT3 PROTEIN; SUPPRESSOR OF CYTOKINE SIGNALING 1; VALINE;

EID: 33845573026     PISSN: 10428194     EISSN: 10292403     Source Type: Journal    
DOI: 10.1080/10428190600925772     Document Type: Letter
Times cited : (4)

References (12)
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  • 2
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  • 3
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  • 4
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    • Role of the STAT1 pathway in apoptosis induced by fludarabine and JAK kinase inhibitors in B-cell chronic lymphocytic leukemia
    • Martinez-Lostao, L and Briones, J and Forne, I and Martinez-Gallo, M and Ferrer, B and Sierra, J and (2005) Role of the STAT1 pathway in apoptosis induced by fludarabine and JAK kinase inhibitors in B-cell chronic lymphocytic leukemia Leuk Lymphoma, 46, pp. 435-442.
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  • 6
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  • 7
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    • The JAK2 V617F activating mutation occurs in chronic myelomonocytic leukemia and acute myeloid leukemia, but not in acute lymphoblastic blastic leukemia or chronic lymphocytic leukemia
    • Levine, R and Loriaux, M and Huntly, B and Loh, M and Beran, M and Stoffregen, E and (2005) The JAK2 V617F activating mutation occurs in chronic myelomonocytic leukemia and acute myeloid leukemia, but not in acute lymphoblastic blastic leukemia or chronic lymphocytic leukemia Blood, 106, pp. 3377-3379.
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  • 8
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    • The JAK2 V617F activating tyrosine kinase mutation is an infrequent event in both 'atypical' myeloproliferative disorders and the myelodysplastic syndrome
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.