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Volumn 93, Issue 3, 2003, Pages
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The antianginal agent trimetazidine does not exert its functional benefit via inhibition of mitochondrial long-chain 3-ketoacyl coenzyme A thiolase.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
ACETYL COENZYME A ACYLTRANSFERASE;
ENZYME INHIBITOR;
PIPERAZINE DERIVATIVE;
RANOLAZINE;
RECOMBINANT PROTEIN;
TRIMETAZIDINE;
VASODILATOR AGENT;
ANIMAL;
ARTICLE;
CELL LINE;
CHEMISTRY;
CYTOLOGY;
DOSE RESPONSE;
DRUG ANTAGONISM;
DRUG EFFECT;
ENZYME ACTIVATION;
ENZYMOLOGY;
HEART;
HEART MITOCHONDRION;
HEART MUSCLE;
HEART MUSCLE CELL;
HEART MUSCLE ISCHEMIA;
HUMAN;
IN VITRO STUDY;
ISOLATION AND PURIFICATION;
MALE;
METABOLISM;
OXIDATION REDUCTION REACTION;
PATHOPHYSIOLOGY;
RAT;
ACETYL-COA C-ACYLTRANSFERASE;
ANIMALS;
CELL LINE;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ENZYME ACTIVATION;
ENZYME INHIBITORS;
HEART;
HUMANS;
MALE;
MITOCHONDRIA, HEART;
MYOCARDIAL ISCHEMIA;
MYOCARDIUM;
MYOCYTES, CARDIAC;
OXIDATION-REDUCTION;
PIPERAZINES;
RATS;
RECOMBINANT PROTEINS;
TRIMETAZIDINE;
VASODILATOR AGENTS;
MLCS;
MLOWN;
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EID: 0141723313
PISSN: None
EISSN: 15244571
Source Type: Journal
DOI: 10.1161/01.res.0000086943.72932.71 Document Type: Article |
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Times cited : (108)
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References (0)
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