Overexpression of α(1B)-adrenergic receptor induces left ventricular dysfunction in the absence of hypertrophy

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 275, Issue 4 44-4, 1998, Pages

  Grupp, Ingrid L ^a   Lorenz, John N ^a   Walsh, Richard A ^b   Boivin, Gregory P ^a   Rindt, Hansjörg ^c  

^a UNIVERSITY OF CINCINNATI   (United States)

^b UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

^c MONTREAL HEART INSTITUTE   (Canada)

Author keywords

Heart; Muscle; Myocardial contractility; Transgenic mouse

Indexed keywords

ALPHA ADRENERGIC STIMULATION; ANIMAL EXPERIMENT; ARTICLE; HEART HYPERTROPHY; HEART LEFT VENTRICLE FAILURE; HEART MUSCLE CONTRACTILITY; INOTROPISM; MOUSE; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN EXPRESSION; RECEPTOR DENSITY; TRANSGENIC MOUSE; ANIMAL; BLOOD PRESSURE; C57BL MOUSE; CARDIOMEGALY; CELL CULTURE; DRUG ANTAGONISM; DRUG EFFECT; GENETICS; HEART; HEART CONTRACTION; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE; HEART RATE; IN VITRO STUDY; METABOLISM; MOUSE STRAIN; PHYSIOLOGY; REGRESSION ANALYSIS; ULTRASTRUCTURE;

ADRENERGIC RECEPTOR ALPHA(1B); ALPHA 1 ADRENERGIC RECEPTOR; CALCIUM; ISOPRENALINE; PRAZOSIN;

ANIMALS; BLOOD PRESSURE; CALCIUM; CARDIOMEGALY; CELLS, CULTURED; HEART; HEART RATE; ISOPROTERENOL; MICE; MICE, INBRED C57BL; MICE, INBRED STRAINS; MICE, TRANSGENIC; MYOCARDIAL CONTRACTION; MYOCARDIUM; PRAZOSIN; RECEPTORS, ADRENERGIC, ALPHA-1; REGRESSION ANALYSIS; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR FUNCTION, LEFT;

EID: 33750889809     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.1998.275.4.h1338     Document Type: Article

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