The activation loop phosphorylation of protein kinase D is an early marker of neuronal DNA damage

Journal of Neurochemistry

Volumn 99, Issue 1, 2006, Pages 218-225

  Besirli, Cagri G ^a   Johnson Jr , Eugene M ^a,b  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b Campus Box 8103 ^*  (United States)

Author keywords

Apoptosis; Ataxia telangiectasia mutated; Neuron; Protein kinase C; Protein kinase C

Indexed keywords

ATM PROTEIN; ETOPOSIDE; PROTEIN KINASE C; PROTEIN KINASE C MU; PROTEIN KINASE D; TOXIC SUBSTANCE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATAXIA TELANGIECTASIA; CELL SURVIVAL; CONTROLLED STUDY; DNA DAMAGE; DOWNSTREAM PROCESSING; ENZYME ACTIVATION; ENZYME PHOSPHORYLATION; GENOTOXICITY; IN VIVO STUDY; INDUCED MUTATION; NERVE CELL; NERVE CELL CULTURE; NERVE CELL LESION; NEURONAL DNA DAMAGE; NEWBORN; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN DOMAIN; RAT; STRESS;

ANIMALS; ANIMALS, NEWBORN; BIOLOGICAL MARKERS; CELL CYCLE PROTEINS; CELLS, CULTURED; DNA DAMAGE; DNA-BINDING PROTEINS; ENZYME ACTIVATION; NEURONS; PHOSPHORYLATION; PROTEIN KINASE C; PROTEIN-SERINE-THREONINE KINASES; RATS; RATS, SPRAGUE-DAWLEY; SYMPATHETIC NERVOUS SYSTEM; TUMOR SUPPRESSOR PROTEINS;

ATAXIA TELANGIECTASIA;

EID: 33748683346     PISSN: 00223042     EISSN: 14714159     Source Type: Journal    
DOI: 10.1111/j.1471-4159.2006.04116.x     Document Type: Article

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