Hypertonic saline dextran after burn injury decreases inflammatory cytokine responses to subsequent pneumonia-related sepsis

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 290, Issue 4, 2006, Pages

  Horton, Jureta W ^a   Maass, David L ^a   White, D Jean ^a  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

Burn complicated by sepsis; Cardiac myocytes; Interleukin 1 ; Interleukin 6; Langendorff perfusion; Rat model; Tumor necrosis factor

Indexed keywords

CALCIUM; CYTOKINE; DEXTRAN; ISOTONIC SOLUTION; RINGER LACTATE SOLUTION; SODIUM; SODIUM CHLORIDE; TUMOR NECROSIS FACTOR ALPHA; HYPERTONIC SALINE DEXTRAN SOLUTION; HYPERTONIC SALINE-DEXTRAN SOLUTION;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BODY SURFACE; BURN; COLONY FORMING UNIT; CONTROLLED STUDY; DRUG EFFECT; HEART CONTRACTION; HEART MUSCLE CELL; HEART MUSCLE RELAXATION; HEMODYNAMIC PARAMETERS; INFLAMMATION; NONHUMAN; PNEUMONIA; PRIORITY JOURNAL; RAT; RESUSCITATION; SEPSIS; STREPTOCOCCUS PNEUMONIAE; ANIMAL; BACTERIAL PNEUMONIA; IMMUNOLOGY; MYOCARDITIS; TREATMENT OUTCOME; WISTAR FURTH RAT;

ANIMALS; BURNS; CYTOKINES; DEXTRANS; MYOCARDIAL CONTRACTION; MYOCARDITIS; PNEUMONIA, STAPHYLOCOCCAL; RATS; RATS, INBRED WF; SODIUM CHLORIDE; TREATMENT OUTCOME;

EID: 33646086980     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00586.2005     Document Type: Article

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