TNF receptor deficiency reveals a translational control mechanism for adriamycin-induced Fas expression in cardiac tissues

Cytokine

Volumn 33, Issue 4, 2006, Pages 226-230

  Lien, Yu Chin ^a   Daosukho, Chotiros ^a,b   St Clair, Daret K ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b MAHIDOL UNIVERSITY   (Thailand)

Author keywords

Adriamycin; AP 1; Fas; p53; TNF receptors

Indexed keywords

DOXORUBICIN; FAS ANTIGEN; MESSENGER RNA; PROTEIN P53; TRANSCRIPTION FACTOR AP 1; TUMOR NECROSIS FACTOR RECEPTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIGEN EXPRESSION; ARTICLE; CARDIOTOXICITY; CONTROLLED STUDY; HEART; MALE; MOUSE; MOUSE STRAIN; NONHUMAN; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; RNA TRANSLATION;

ANIMALS; ANTIBIOTICS, ANTINEOPLASTIC; ANTIGENS, CD95; DOXORUBICIN; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PROTEIN BIOSYNTHESIS; RECEPTORS, TUMOR NECROSIS FACTOR, TYPE I; RECEPTORS, TUMOR NECROSIS FACTOR, TYPE II; TRANSCRIPTION FACTOR AP-1; TUMOR SUPPRESSOR PROTEIN P53;

ANIMALIA;

EID: 33646036744     PISSN: 10434666     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cyto.2006.02.001     Document Type: Article

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