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Annals of the New York Academy of Sciences
Volumn 1019, Issue , 2004, Pages 274-277
Investigation of the signaling pathways involved in the proliferative life span barriers in werner syndrome fibroblasts
Author keywords

Aging; Cyclin dependent kinases; Oncoproteins; p16 INK4a; p21WAF1; p53; Replicative senescence; Telomerase; Telomeres
Indexed keywords

ONCOPROTEIN; PROTEIN P16; PROTEIN P21; PROTEIN P53;
EID: 3242739250     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1196/annals.1297.046     Document Type: Conference Paper
Times cited : (8)
References (10)
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    • FARAGHER, R.G.A., I.R. KILL, J.A. HUNTER, et al. 1993. The gene responsible for Werner syndrome may be a cell division "counting" gene. Proc. Natl. Acad. Sci. USA 90: 12030-12034.
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    • WYLLIE, F.S., C.J. JONES, J.W. SKINNER, et al. 2000. Telomerase prevents the accelerated ageing of Werner syndrome fibroblasts. Nat. Genet. 24: 16-17.
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    • Control of replicative life span in human cells: Barriers to clonal expansion intermediate between M1 senescence and M2 crisis
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    • Telomere-based proliferative life span barriers in Werner syndrome fibroblasts involve both p53-dependent and p53-independent mechanisms
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.