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Volumn 13, Issue 2, 2006, Pages 348-351
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HIV-1 protease inhibitors and cytomegalovirus vMIA induce mitochondrial fragmentation without triggering apoptosis [2]
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Author keywords
[No Author keywords available]
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Indexed keywords
CALCIUM ION;
HEAT SHOCK PROTEIN 60;
INHIBITOR OF APOPTOSIS PROTEIN;
NELFINAVIR;
PROTEINASE INHIBITOR;
RITONAVIR;
STAUROSPORINE;
APOPTOSIS;
CALCIUM CELL LEVEL;
CALCIUM TRANSPORT;
CELL DIVISION;
CELL MEMBRANE PERMEABILITY;
CELL MEMBRANE POTENTIAL;
CELL VIABILITY;
CELLULAR DISTRIBUTION;
CYTOMEGALOVIRUS;
DRUG MECHANISM;
HEPATITIS;
HIGHLY ACTIVE ANTIRETROVIRAL THERAPY;
HUMAN;
HUMAN IMMUNODEFICIENCY VIRUS 1;
HUMAN IMMUNODEFICIENCY VIRUS INFECTION;
IN VITRO STUDY;
LETTER;
MITOCHONDRIAL MEMBRANE;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN LOCALIZATION;
APOPTOSIS;
CALCIUM;
CERAMIDES;
CYTOMEGALOVIRUS;
GIANT CELLS;
HELA CELLS;
HIV PROTEASE INHIBITORS;
HUMANS;
HYDROGEN PEROXIDE;
IMMEDIATE-EARLY PROTEINS;
INHIBITOR OF APOPTOSIS PROTEINS;
MACROPHAGE INFLAMMATORY PROTEINS;
MITOCHONDRIA;
NELFINAVIR;
RITONAVIR;
SAQUINAVIR;
TUMOR SUPPRESSOR PROTEIN P53;
VIRAL PROTEINS;
CYTOMEGALOVIRUS;
HUMAN IMMUNODEFICIENCY VIRUS;
HUMAN IMMUNODEFICIENCY VIRUS 1;
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EID: 30844440999
PISSN: 13509047
EISSN: 14765403
Source Type: Journal
DOI: 10.1038/sj.cdd.4401750 Document Type: Letter |
Times cited : (22)
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References (35)
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