Increased Neuronal Firing in Computer Simulations of Sodium Channel Mutations That Cause Generalized Epilepsy with Febrile Seizures Plus

Journal of Neurophysiology

Volumn 91, Issue 5, 2004, Pages 2040-2050

  Spampanato, Jay ^a   Aradi, Ildiko ^b   Soltesz, Ivan ^b   Goldin, Alan L ^a,c  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

4 AMINOBUTYRIC ACID A RECEPTOR; GENE PRODUCT; POTASSIUM CHANNEL; PROTEIN SCN1A; PROTEIN SCN1B; PROTEIN SCN2A; SODIUM CHANNEL; UNCLASSIFIED DRUG;

ACTION POTENTIAL; ARTICLE; AUTOSOMAL DOMINANT DISORDER; CHANNEL GATING; COMPUTER SIMULATION; EPILEPTOGENESIS; FEBRILE CONVULSION; GENE ACTIVATION; GENE INACTIVATION; GENE MUTATION; GENERALIZED EPILEPSY; GENETIC CODE; PHENOTYPE; PRIORITY JOURNAL;

ACTION POTENTIALS; ALGORITHMS; ANIMALS; COMPUTER SIMULATION; ELECTROPHYSIOLOGY; EPILEPSY, GENERALIZED; FEVER; KINETICS; MODELS, NEUROLOGICAL; MUTATION; NEURONS; OOCYTES; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS, INWARDLY RECTIFYING; SEIZURES; SODIUM CHANNEL BLOCKERS; SODIUM CHANNELS; SOFTWARE; XENOPUS LAEVIS;

EID: 3042792173     PISSN: 00223077     EISSN: None     Source Type: Journal    
DOI: 10.1152/jn.00982.2003     Document Type: Article

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