Microarray expression analysis of gad mice implicates involvement of Parkinson's disease associated UCH-L1 in multiple metabolic pathways

Molecular Brain Research

Volumn 126, Issue 1, 2004, Pages 88-97

  Bonin, M ^a   Poths, S ^a   Osaka, H ^b   Wang, Y L ^b   Wada, K ^b   Riess, O ^a  

^a UNIVERSITY OF TÜBINGEN   (Germany)

^b NATIONAL INSTITUTE OF NEUROSCIENCE   (Japan)

Author keywords

Synuclein; Disorders of the nervous system; Expression; Genetic models; Microarray chip analysis; Parkinson's disease; Protein degradation; Ubiquitin; UCH L1

Indexed keywords

ALPHA SYNUCLEIN; BRAIN DERIVED NEUROTROPHIC FACTOR; CCAAT BINDING FACTOR; GENE PRODUCT; PARKIN; PROTEIN SUBUNIT; UBIQUITIN C TERMINAL HYDROLASE L1; UBIQUITIN THIOLESTERASE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DNA MICROARRAY; DOWN REGULATION; GENE CONTROL; GENE EXPRESSION PROFILING; GENE MUTATION; GENETIC ASSOCIATION; MALE; MOUSE; NERVE FIBER DEGENERATION; NEUROPATHOLOGY; NONHUMAN; NUCLEOTIDE SEQUENCE; PARKINSON DISEASE; PRIORITY JOURNAL; PROTEIN DEGRADATION; SPECIES COMPARISON; UNINDEXED SEQUENCE; UPREGULATION; WILD TYPE;

ANIMALS; BRAIN; CCAAT-ENHANCER-BINDING PROTEINS; CLUSTER ANALYSIS; GENE EXPRESSION PROFILING; GENE EXPRESSION REGULATION; HUMANS; MALE; MICE; MICE, MUTANT STRAINS; MOLECULAR SEQUENCE DATA; OLIGONUCLEOTIDE ARRAY SEQUENCE ANALYSIS; PARKINSON DISEASE; UBIQUITIN THIOLESTERASE;

ANIMALIA;

EID: 2942635265     PISSN: 0169328X     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.molbrainres.2004.03.025     Document Type: Article

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