-
1
-
-
0031750294
-
Basic and clinical aspects of myocardial stunning
-
Bolli R. Basic and clinical aspects of myocardial stunning. Prog Cardiovasc Dis 40: 477-516, 1998.
-
(1998)
Prog Cardiovasc Dis
, vol.40
, pp. 477-516
-
-
Bolli, R.1
-
2
-
-
0023219426
-
Evidence for a pathogenetic role of xanthine oxidase in the "stunned" myocardium
-
Charlat MI, O'Neill PG, Egan JM, Abernethy DR, Michael LH, Myers ML, Roberts R, and Bolli R. Evidence for a pathogenetic role of xanthine oxidase in the "stunned" myocardium. Am J Physiol Heart Circ Physiol 252: H566-H577, 1987.
-
(1987)
Am J Physiol Heart Circ Physiol
, vol.252
-
-
Charlat, M.I.1
O'Neill, P.G.2
Egan, J.M.3
Abernethy, D.R.4
Michael, L.H.5
Myers, M.L.6
Roberts, R.7
Bolli, R.8
-
3
-
-
0035818566
-
Effects of nitric oxide synthase inhibition on basal function and the force-frequency relationship in the normal and failing human heart in vivo
-
Cotton JM, Kearney MT, MacCarthy PA, Grocott-Mason RM, McClean DR, Heymes C, Richardson PJ, and Shah AM. Effects of nitric oxide synthase inhibition on basal function and the force-frequency relationship in the normal and failing human heart in vivo. Circulation 104: 2318-2323, 2001.
-
(2001)
Circulation
, vol.104
, pp. 2318-2323
-
-
Cotton, J.M.1
Kearney, M.T.2
MacCarthy, P.A.3
Grocott-Mason, R.M.4
McClean, D.R.5
Heymes, C.6
Richardson, P.J.7
Shah, A.M.8
-
4
-
-
0028356959
-
Antioxidant changes in hypertrophied and failing guinea pig hearts
-
Dhalla AK and Singal PK. Antioxidant changes in hypertrophied and failing guinea pig hearts. Am J Physiol Heart Circ Physiol 266: H1280-H1285, 1994.
-
(1994)
Am J Physiol Heart Circ Physiol
, vol.266
-
-
Dhalla, A.K.1
Singal, P.K.2
-
5
-
-
0033520357
-
Intravenous allopurinol decreases myocardial oxygen consumption and increases mechanical efficiency in dogs with pacing-induced heart failure
-
Ekelund UE, Harrison RW, Shokek O, Thakkar RN, Tunin RS, Senzaki H, Kass DA, Marban E, and Hare JM. Intravenous allopurinol decreases myocardial oxygen consumption and increases mechanical efficiency in dogs with pacing-induced heart failure. Circ Res 85: 437-445, 1999.
-
(1999)
Circ Res
, vol.85
, pp. 437-445
-
-
Ekelund, U.E.1
Harrison, R.W.2
Shokek, O.3
Thakkar, R.N.4
Tunin, R.S.5
Senzaki, H.6
Kass, D.A.7
Marban, E.8
Hare, J.M.9
-
6
-
-
0037080553
-
Role of myocardial inducible nitric oxide synthase in contractile dysfunction and β-adrenergic hyporesponsiveness in rats with experimental volume-overload heart failure
-
Gealekman O, Abassi Z, Rubinstein I, Winaver J, and Binah O. Role of myocardial inducible nitric oxide synthase in contractile dysfunction and β-adrenergic hyporesponsiveness in rats with experimental volume-overload heart failure. Circulation 105: 236-243, 2002.
-
(2002)
Circulation
, vol.105
, pp. 236-243
-
-
Gealekman, O.1
Abassi, Z.2
Rubinstein, I.3
Winaver, J.4
Binah, O.5
-
7
-
-
0035852761
-
Antioxidants and myocardial contractility: Illuminating the "dark side" of β-adrenergic receptor activation?
-
Givertz MM, Sawyer DB, and Colucci WS. Antioxidants and myocardial contractility: illuminating the "dark side" of β-adrenergic receptor activation? Circulation 103: 782-783.
-
Circulation
, vol.103
, pp. 782-783
-
-
Givertz, M.M.1
Sawyer, D.B.2
Colucci, W.S.3
-
8
-
-
0024308421
-
Effects of exogenous free radicals on electromechanical function and metabolism in isolated rabbit and guinea pig ventricle
-
Goldhaber JI, Ji S, Lamp ST, and Weiss JN. Effects of exogenous free radicals on electromechanical function and metabolism in isolated rabbit and guinea pig ventricle. J Clin Invest 83: 1800-1809, 1989.
-
(1989)
J Clin Invest
, vol.83
, pp. 1800-1809
-
-
Goldhaber, J.I.1
Ji, S.2
Lamp, S.T.3
Weiss, J.N.4
-
9
-
-
0022640297
-
Superoxide anion is involved in the breakdown of endothelium-derived vascular relaxing factor
-
Gryglewski RJ, Palmer RM, and Moncada S. Superoxide anion is involved in the breakdown of endothelium-derived vascular relaxing factor. Nature 320: 454-456, 1986.
-
(1986)
Nature
, vol.320
, pp. 454-456
-
-
Gryglewski, R.J.1
Palmer, R.M.2
Moncada, S.3
-
10
-
-
0031917409
-
Increased sensitivity to nitric oxide synthase inhibition in patients with heart failure. Potentiation of β-adrenergic inotropic responsiveness
-
Hare JM, Givertz MM, Creager MA, and Colucci WS. Increased sensitivity to nitric oxide synthase inhibition in patients with heart failure. Potentiation of β-adrenergic inotropic responsiveness. Circulation 97: 161-166, 1998.
-
(1998)
Circulation
, vol.97
, pp. 161-166
-
-
Hare, J.M.1
Givertz, M.M.2
Creager, M.A.3
Colucci, W.S.4
-
11
-
-
0028844217
-
Nitric oxide inhibits the positive inotropic response to β-adrenergic stimulation in humans with left ventricular dysfunction
-
Hare JM, Loh E, Creager MA, and Colucci WS. Nitric oxide inhibits the positive inotropic response to β-adrenergic stimulation in humans with left ventricular dysfunction. Circulation 92: 2198-2203, 1995.
-
(1995)
Circulation
, vol.92
, pp. 2198-2203
-
-
Hare, J.M.1
Loh, E.2
Creager, M.A.3
Colucci, W.S.4
-
12
-
-
0037785375
-
Nitric oxide and excitation-contraction coupling
-
Hare JM. Nitric oxide and excitation-contraction coupling. J Mol Cell Cardiol 35: 719-729, 2003.
-
(2003)
J Mol Cell Cardiol
, vol.35
, pp. 719-729
-
-
Hare, J.M.1
-
13
-
-
8344233331
-
Nitroso-redox balance in the cardiovascular system
-
Hare JM. Nitroso-redox balance in the cardiovascular system. N Engl J Med 351: 2112-2114, 2004.
-
(2004)
N Engl J Med
, vol.351
, pp. 2112-2114
-
-
Hare, J.M.1
-
14
-
-
0032476697
-
Ascorbate prevents the interaction of superoxide and nitric oxide only at very high physiological concentrations
-
Jackson TS, Xu A, Vita JA, and Keaney JF. Ascorbate prevents the interaction of superoxide and nitric oxide only at very high physiological concentrations. Circ Res 83: 916-922, 1998.
-
(1998)
Circ Res
, vol.83
, pp. 916-922
-
-
Jackson, T.S.1
Xu, A.2
Vita, J.A.3
Keaney, J.F.4
-
17
-
-
0033947365
-
Unsaturated aldehydes including 4-OH-nonenal are elevated in patients with congestive heart failure
-
Mak S, Lehotay DC, Yazdanpanah M, Azevedo ER, Liu PP, and Newton GE. Unsaturated aldehydes including 4-OH-nonenal are elevated in patients with congestive heart failure. J Card Fail 6: 108-114, 2000.
-
(2000)
J Card Fail
, vol.6
, pp. 108-114
-
-
Mak, S.1
Lehotay, D.C.2
Yazdanpanah, M.3
Azevedo, E.R.4
Liu, P.P.5
Newton, G.E.6
-
18
-
-
0035852701
-
Vitamin C augments the inotropic response to dobutamine in humans with normal left ventricular function
-
Mak S and Newton GE. Vitamin C augments the inotropic response to dobutamine in humans with normal left ventricular function. Circulation 103: 826-830, 2001.
-
(2001)
Circulation
, vol.103
, pp. 826-830
-
-
Mak, S.1
Newton, G.E.2
-
19
-
-
0035680903
-
The oxidative stress hypothesis of congestive heart failure: Radical thoughts
-
Mak S and Newton GE. The oxidative stress hypothesis of congestive heart failure: radical thoughts. Chest 120: 2035-2046.
-
Chest
, vol.120
, pp. 2035-2046
-
-
Mak, S.1
Newton, G.E.2
-
20
-
-
0942265667
-
Redox modulation of the inotropic response to dobutamine is impaired in patients with heart failure
-
Mak S and Newton GE. Redox modulation of the inotropic response to dobutamine is impaired in patients with heart failure. Am J Physiol Heart Circ Physiol 286: H789-H795, 2004.
-
(2004)
Am J Physiol Heart Circ Physiol
, vol.286
-
-
Mak, S.1
Newton, G.E.2
-
21
-
-
0042917516
-
Nitric oxide and cardiac function: Ten years after, and continuing
-
Massion PB, Feron O, Dessy C, and Balligand JL. Nitric oxide and cardiac function: ten years after, and continuing. Circ Res 93: 388-398, 2003.
-
(2003)
Circ Res
, vol.93
, pp. 388-398
-
-
Massion, P.B.1
Feron, O.2
Dessy, C.3
Balligand, J.L.4
-
22
-
-
0034190373
-
How does ascorbic acid prevent endothelial dysfunction?
-
May JM. How does ascorbic acid prevent endothelial dysfunction? Free Rad Biol Med 28: 1421-1429, 2000.
-
(2000)
Free Rad Biol Med
, vol.28
, pp. 1421-1429
-
-
May, J.M.1
-
23
-
-
0031844699
-
Nitric oxide synthases: Catalytic function and progress towards selective inhibition
-
Mayer B and Andrew P. Nitric oxide synthases: catalytic function and progress towards selective inhibition. Arch Pharmacol 358: 127-133, 1998.
-
(1998)
Arch Pharmacol
, vol.358
, pp. 127-133
-
-
Mayer, B.1
Andrew, P.2
-
24
-
-
0031080272
-
β-adrenoceptor-linked signal transduction in ischemic-reperfused heart and scavenging of oxyradicals
-
Persad S, Takeda S, Panza JA, and Dhalla NS. β-Adrenoceptor-linked signal transduction in ischemic-reperfused heart and scavenging of oxyradicals. J Mol Cell Cardiol 29: 545-558, 1997.
-
(1997)
J Mol Cell Cardiol
, vol.29
, pp. 545-558
-
-
Persad, S.1
Takeda, S.2
Panza, J.A.3
Dhalla, N.S.4
-
25
-
-
0030886167
-
Biphasic alterations in cardiac β-adrenoceptor signal transduction mechanism due to oxyradicals
-
Persad S, Elimban V, Kaila J, and Dhalla NS. Biphasic alterations in cardiac β-adrenoceptor signal transduction mechanism due to oxyradicals. J Pharmacol Exp Ther 282: 1623-1631, 1997.
-
(1997)
J Pharmacol Exp Ther
, vol.282
, pp. 1623-1631
-
-
Persad, S.1
Elimban, V.2
Kaila, J.3
Dhalla, N.S.4
-
26
-
-
0037155054
-
Imbalance between xanthine oxidase and nitric oxide synthase signaling pathways underlies mechanoenergetic uncoupling in the failing heart
-
Saavedra WF, Paolocci N, St John ME, Skaf MW, Stewart GC, Xie JS, Harrison RW, Zeichner J, Mudrick D, Marban E, Kass DA, and Hare JM. Imbalance between xanthine oxidase and nitric oxide synthase signaling pathways underlies mechanoenergetic uncoupling in the failing heart. Circ Res 90: 297-304.
-
Circ Res
, vol.90
, pp. 297-304
-
-
Saavedra, W.F.1
Paolocci, N.2
St. John, M.E.3
Skaf, M.W.4
Stewart, G.C.5
Xie, J.S.6
Harrison, R.W.7
Zeichner, J.8
Mudrick, D.9
Marban, E.10
Kass, D.A.11
Hare, J.M.12
-
27
-
-
0023687893
-
Quantitative identification of superoxide anion as a negative inotropic species
-
Schrier GM and Hess ML. Quantitative identification of superoxide anion as a negative inotropic species. Am J Physiol Heart Circ Physiol 255: H138-H143, 1988.
-
(1988)
Am J Physiol Heart Circ Physiol
, vol.255
-
-
Schrier, G.M.1
Hess, M.L.2
-
28
-
-
0034033581
-
Pharmacological concentrations of ascorbic acid are required for the beneficial effect on endothelial vasomotor function in hypertension
-
Sherman DL, Keaney JF, Biegelson ES, Duffy SJ, Coffman JD, and Vita JA. Pharmacological concentrations of ascorbic acid are required for the beneficial effect on endothelial vasomotor function in hypertension. Hypertension 35: 936-941, 2000.
-
(2000)
Hypertension
, vol.35
, pp. 936-941
-
-
Sherman, D.L.1
Keaney, J.F.2
Biegelson, E.S.3
Duffy, S.J.4
Coffman, J.D.5
Vita, J.A.6
-
29
-
-
0034712641
-
Nitric oxide spares myocardial oxygen consumption through attenuation of contractile response to β-adrenergic stimulation in patients with idiopathic dilated cardiomyopathy
-
Shinke T, Takaoka H, Takeuchi M, Hata K, Kawai H, Okubo H, Kijima Y, Murata T, and Yokoyama M. Nitric oxide spares myocardial oxygen consumption through attenuation of contractile response to β-adrenergic stimulation in patients with idiopathic dilated cardiomyopathy. Circulation 101: 1925-1930.
-
Circulation
, vol.101
, pp. 1925-1930
-
-
Shinke, T.1
Takaoka, H.2
Takeuchi, M.3
Hata, K.4
Kawai, H.5
Okubo, H.6
Kijima, Y.7
Murata, T.8
Yokoyama, M.9
-
30
-
-
0035929149
-
Nitrosylation. The prototypic redox-based signaling mechanism
-
Stamler JS, Lamas S, and Fang FC. Nitrosylation. The prototypic redox-based signaling mechanism. Cell 106: 675-683, 2001.
-
(2001)
Cell
, vol.106
, pp. 675-683
-
-
Stamler, J.S.1
Lamas, S.2
Fang, F.C.3
-
31
-
-
0034623864
-
Role of endothelium-derived nitric oxide in the regulation of cardiac oxygen metabolism: Implications in health and disease
-
Trochu JN, Bouhour JB, Kaley G, and Hintze TH. Role of endothelium-derived nitric oxide in the regulation of cardiac oxygen metabolism: implications in health and disease. Circ Res 87: 1108-1117, 2000.
-
(2000)
Circ Res
, vol.87
, pp. 1108-1117
-
-
Trochu, J.N.1
Bouhour, J.B.2
Kaley, G.3
Hintze, T.H.4
-
32
-
-
0035814786
-
Allopurinol enhances the contractile response to dobutamine and exercise in dogs with pacing-induced heart failure
-
Ukai T, Cheng CP, Tachibana H, Igawa A, Zhang ZS, Cheng HJ, and Little WC. Allopurinol enhances the contractile response to dobutamine and exercise in dogs with pacing-induced heart failure. Circulation 103: 750-755, 2001.
-
(2001)
Circulation
, vol.103
, pp. 750-755
-
-
Ukai, T.1
Cheng, C.P.2
Tachibana, H.3
Igawa, A.4
Zhang, Z.S.5
Cheng, H.J.6
Little, W.C.7
-
34
-
-
0034890764
-
Cardiac nitric oxide production due to angiotensin-converting enzyme inhibition decreases β-adrenergic myocardial contractility in patients with dilated cardiomyopathy
-
Wittstein IS, Kass DA, Pak PH, Maughan WL, Fetics B, and Hare JM. Cardiac nitric oxide production due to angiotensin-converting enzyme inhibition decreases β-adrenergic myocardial contractility in patients with dilated cardiomyopathy. J Am Coll Cardiol 38: 429-435.
-
J Am Coll Cardiol
, vol.38
, pp. 429-435
-
-
Wittstein, I.S.1
Kass, D.A.2
Pak, P.H.3
Maughan, W.L.4
Fetics, B.5
Hare, J.M.6
-
35
-
-
0031015476
-
2+-ATPase function by direct attack on the ATP binding site
-
2+-ATPase function by direct attack on the ATP binding site. Circ Res 80: 76-81, 1997.
-
(1997)
Circ Res
, vol.80
, pp. 76-81
-
-
Xu, K.Y.1
Zweier, J.L.2
Becker, L.C.3
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