Acetylcholine and sodium nitroprusside cause long-term inhibition of EDCF-mediated contractions

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 289, Issue 6 58-6, 2005, Pages

  Tang, Eva H C ^a   Feletou, Michel ^b   Huang, Yu ^c   Man, Ricky Y K ^a   Vanhoutte, Paul M ^a  

^a UNIVERSITY OF HONG KONG   (Hong Kong)

^b INSTITUT DE RECHERCHES INTERNATIONALES SERVIER   (France)

^c and Hong Kong Branch of CAS Center for Excellence in Animal Evolution and Genetics   (Hong Kong)

Author keywords

Endothelium dependent contractions; Endothelium derived contracting factors; Nitric oxide; Spontaneously hypertensive rats

Indexed keywords

15 HYDROXY 11ALPHA,9ALPHA EPOXYMETHANOPROSTA 5,13 DIENOIC ACID; 1H 1,2,4 OXADIAZOLO[4,3 A]QUINOXALIN 1 ONE; ACETYLCHOLINE; CALCIMYCIN; ENDOTHELIUM DERIVED CONSTRICTING FACTOR; GUANYLATE CYCLASE INHIBITOR; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE SYNTHASE INHIBITOR; NITROPRUSSIDE SODIUM; PHENYLEPHRINE; THROMBOXANE;

ANIMAL TISSUE; AORTA CONSTRICTION; ARTICLE; CALCIUM BLOOD LEVEL; CONCENTRATION RESPONSE; CONTROLLED STUDY; MALE; MUSCLE FORCE; MUSCLE ISOMETRIC CONTRACTION; NONHUMAN; PRIORITY JOURNAL; RAT; SPONTANEOUSLY HYPERTENSIVE RAT; VASCULAR ENDOTHELIUM;

ACETYLCHOLINE; ADAPTATION, PHYSIOLOGICAL; ANIMALS; DOSE-RESPONSE RELATIONSHIP, DRUG; DRUG COMBINATIONS; ENDOTHELINS; HYPERTENSION; MALE; MYOCARDIAL CONTRACTION; NITROPRUSSIDE; RATS; RATS, INBRED SHR; TIME FACTORS;

EID: 28144431646     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00568.2005     Document Type: Article

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