Major histocompatibility complex class II molecules can protect from diabetes by positively selecting T cells with additional specificities

Journal of Experimental Medicine

Volumn 187, Issue 3, 1998, Pages 379-387

  Lühder, Fred ^a   Katz, Jonathan ^a,b   Benoist, Christophe ^a   Mathis, Diane ^a  

^a INSTITUT DE GÉNÉTIQUE ET DE BIOLOGIE MOLÉCULAIRE ET CELLULAIRE   (France)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2; T LYMPHOCYTE RECEPTOR;

ALLELE; ANIMAL CELL; ANTIGEN PRESENTING CELL; ARTICLE; GENE REARRANGEMENT; HUMAN; HYBRIDOMA; INSULIN DEPENDENT DIABETES MELLITUS; INSULITIS; MAJOR HISTOCOMPATIBILITY COMPLEX; MOUSE; PATHOGENESIS; PENETRANCE; PRIORITY JOURNAL; TRANSGENIC ANIMAL;

ANIMALS; ANTIGEN-PRESENTING CELLS; ANTIGENS, CD; DIABETES MELLITUS, TYPE 1; DISEASE MODELS, ANIMAL; GENES, MHC CLASS II; HAPLOTYPES; HEMOCYANIN; HYBRIDOMAS; INTERLEUKIN-2; ISLETS OF LANGERHANS; MICE; MICE, INBRED NOD; MICE, TRANSGENIC; MURAMIDASE; RECEPTORS, ANTIGEN, T-CELL; T-LYMPHOCYTES; TRANSGENES;

EID: 2642654250     PISSN: 00221007     EISSN: None     Source Type: Journal    
DOI: 10.1084/jem.187.3.379     Document Type: Article

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