Inhibition of PKC phosphorylation of cTnI improves cardiac performance in vivo

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 286, Issue 6 55-6, 2004, Pages

  Roman, Brian B ^a   Goldspink, Paul H ^b   Spaite, Elyse ^b   Urboniene, Dalia ^b   McKinney, Ron ^b   Geenen, David L ^b   Solaro, R John ^b   Buttrick, Peter M ^b  

^a UNIVERSITY OF CHICAGO   (United States)

^b NONE

Author keywords

Contractility; Mouse; Protein kinase C; Troponin

Indexed keywords

ALANINE; CELL PROTEIN; CYCLIC AMP; ISOPRENALINE; PHENYLEPHRINE; PROPRANOLOL; PROTEIN KINASE C; PROTEIN KINASE C EPSILON; SERINE; TROPONIN I;

AMINO ACID SUBSTITUTION; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BINDING SITE; CONTROLLED STUDY; ENZYME PHOSPHORYLATION; HEART MUSCLE CONTRACTILITY; HEART MUSCLE DP-DT; HEART PERFORMANCE; HEMODYNAMICS; IN VIVO STUDY; INOTROPISM; MALE; MOUSE; MYOFILAMENT; NONHUMAN; PRESSURE MEASUREMENT; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN PHOSPHORYLATION;

ANIMALS; CALCIUM; CARDIOMEGALY; CARDIOTONIC AGENTS; CORONARY CIRCULATION; CYCLIC AMP; CYCLIC AMP-DEPENDENT PROTEIN KINASES; ECHOCARDIOGRAPHY; ISOPROTERENOL; MALE; MICE; MICE, TRANSGENIC; MUTAGENESIS; MYOCARDIAL CONTRACTION; PHOSPHORYLATION; PROTEIN KINASE C; PROTEIN KINASE C-EPSILON; TROPONIN I;

EID: 2542498796     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00582.2003     Document Type: Article

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