A TASK3 channel (KCNK9) mutation in a genetic model of absence epilepsy

Journal of Molecular Neuroscience

Volumn 25, Issue 1, 2005, Pages 37-51

  Holter, Jethro ^a,c   Carter, David ^a   Leresche, Nathalie ^b   Crunelli, Vincenzo ^a   Vincent, Pierre ^b  

^a CARDIFF UNIVERSITY   (United Kingdom)

^b Groupe Enzymologie Moléculaire et Fonctionnelle   (France)

^c UNIVERSITY OF OSLO   (Norway)

Author keywords

Absence epilepsy; Muscarinic response; pH; Potassium channels; Thalamus

Indexed keywords

ALANINE; BARIUM; DIMER; ISOPROTEIN; MUSCARINIC RECEPTOR; MUTANT PROTEIN; POTASSIUM CHANNEL; PROTEIN; PROTEIN KCNK9; RUTHENIUM RED; UNCLASSIFIED DRUG; COLORING AGENT; KCNK9 PROTEIN, HUMAN; KCNK9 PROTEIN, RAT; TANDEM PORE DOMAIN POTASSIUM CHANNEL;

ABSENCE EPILEPSY; ACIDITY; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN CORTEX; BRAIN NERVE CELL; BRAIN SLICE; CARBOXY TERMINAL SEQUENCE; CHO CELL; CONCENTRATION RESPONSE; CONTROLLED STUDY; EMBRYO; EPILEPSY; FEMALE; GENE MUTATION; GENE SEQUENCE; GENETIC DISORDER; GENETIC MODEL; HUMAN; HUMAN CELL; INTRACELLULAR SPACE; NONHUMAN; NUCLEOTIDE SEQUENCE; PH; POTASSIUM CURRENT; PROTEIN DOMAIN; PROTEIN EXPRESSION; RAT; RAT STRAIN; THALAMUS; TWIK LIKE ACID SENSITIVE POTASSIUM CHANNEL 3; WILD TYPE; ABSENCE; AMINO ACID SEQUENCE; ANIMAL; BIOLOGICAL MODEL; BRAIN; CELL LINE; CHILD; CYTOLOGY; DISEASE MODEL; GENETICS; METABOLISM; MOLECULAR GENETICS; MUTATION; NERVE CELL; PATCH CLAMP; PATHOPHYSIOLOGY; SEQUENCE ALIGNMENT;

RATTUS NORVEGICUS;

AMINO ACID SEQUENCE; ANIMALS; BRAIN; CELL LINE; CHILD; COLORING AGENTS; DISEASE MODELS, ANIMAL; EPILEPSY, ABSENCE; HUMANS; HYDROGEN-ION CONCENTRATION; MODELS, GENETIC; MOLECULAR SEQUENCE DATA; MUTATION; NEURONS; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS, TANDEM PORE DOMAIN; RATS; RATS, INBRED STRAINS; RECEPTORS, MUSCARINIC; RUTHENIUM RED; SEQUENCE ALIGNMENT;

EID: 22344442835     PISSN: 08958696     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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