A functional variant in FCRL3, encoding Fc receptor-like 3, is associated with rheumatoid arthritis and several autoimmunities

Nature Genetics

Volumn 37, Issue 5, 2005, Pages 478-485

  Kochi, Yuta ^a,b   Yamada, Ryo ^a   Suzuki, Akari ^a   Harley, John B ^c   Shirasawa, Senji ^d   Sawada, Tetsuji ^b   Bae, Sang Cheol ^e   Tokuhiro, Shinya ^a   Chang, Xiaotian ^a   Sekine, Akihiro ^a   Takahashi, Atsushi ^a   Tsunoda, Tatsuhiko ^a   Ohnishi, Yozo ^a   Kaufman, Kenneth M ^c   Kang, Changsoo Paul ^f   Kang, Changwon ^f   Otsubo, Shigeru ^g   Yumura, Wako ^h   Mimori, Akio ^d   Koike, Takao ⁱ   Nakamura, Yusuke ^a,g   Sasazuki, Takehiko ^d   Yamamoto, Kazuhiko ^a,b   more..

^a RIKEN   (Japan)

^b UNIVERSITY OF TOKYO   (Japan)

^c UNIVERSITY OF OKLAHOMA   (United States)

^d NATIONAL CENTER FOR GLOBAL HEALTH AND MEDICINE   (Japan)

^e Hanyang Institute of Bioscience and Biotechnology   (South Korea)

^f Korea Advanced Institute of Science and Technology (KAIST)   (South Korea)

^g UNIVERSITY OF TOKYO   (Japan)

^h TOKYO WOMEN'S MEDICAL UNIVERSITY   (Japan)

ⁱ HOKKAIDO UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; FC RECEPTOR; FC RECEPTOR LIKE 3 PROTEIN; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; UNCLASSIFIED DRUG;

ADULT; AGED; ANTIBODY PRODUCTION; ARTICLE; AUTOIMMUNE DISEASE; AUTOIMMUNITY; B LYMPHOCYTE; BINDING AFFINITY; CONTROLLED STUDY; FCRL3 GENE; FEMALE; GENE; GENE EXPRESSION; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; HUMAN; HUMAN CELL; HUMAN TISSUE; MAJOR CLINICAL STUDY; MALE; NORMAL HUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN FAMILY; RHEUMATOID ARTHRITIS; SINGLE NUCLEOTIDE POLYMORPHISM; SYSTEMIC LUPUS ERYTHEMATOSUS;

ARTHRITIS, RHEUMATOID; AUTOIMMUNITY; CASE-CONTROL STUDIES; CHROMOSOMES, HUMAN, PAIR 1; GENE EXPRESSION REGULATION; HLA-DR ANTIGENS; HUMANS; LINKAGE DISEQUILIBRIUM; MOLECULAR SEQUENCE DATA; MULTIGENE FAMILY; MUTATION; POLYMORPHISM, SINGLE NUCLEOTIDE; RECEPTORS, IMMUNOLOGIC;

EID: 20944434679     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/ng1540     Document Type: Article

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