Significance and mechanism of Alzheimer neurofibrillary degeneration and therapeutic targets to inhibit this lesion

Journal of Molecular Neuroscience

Volumn 19, Issue 1-2, 2002, Pages 95-99

  Iqbal, K ^a   Alonso, A d C ^a   El Akkad, E ^a   Gong, C X ^a   Haque, N ^a   Khatoon, S ^a   Pei, J J ^b   Tsujio, I ^a   Wang, J Z ^a,c   Grundke Iqbal, I ^a  

^a NEW YORK STATE INSTITUTE FOR BASIC RESEARCH IN DEVELOPMENTAL DISABILITIES   (United States)

^b KAROLINSKA INSTITUTE   (Sweden)

^c HUAZHONG UNIVERSITY OF SCIENCE AND TECHNOLOGY   (China)

Author keywords

Abnormal hyperphosphorylation; Alzheimer disease; Microtubule associated protein tau; Neurofibrillary tangles; Protein phosphatase 2A

Indexed keywords

MICROTUBULE ASSOCIATED PROTEIN 1; MICROTUBULE ASSOCIATED PROTEIN 2; PHOSPHOPROTEIN PHOSPHATASE; PHOSPHOPROTEIN PHOSPHATASE 2A; PROTEIN KINASE; TAU PROTEIN;

ALZHEIMER DISEASE; ARTICLE; DEPHOSPHORYLATION; DRUG SCREENING; HUMAN; MUTATION; NERVE DEGENERATION; NEUROFIBRILLARY TANGLE; PROTEIN PHOSPHORYLATION; REGULATORY MECHANISM;

EID: 18544370897     PISSN: 08958696     EISSN: None     Source Type: Journal    
DOI: 10.1007/s12031-002-0017-3     Document Type: Article

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