Different presentation of bone mass in mice and humans with congenital leptin deficiency [5]

Journal of Clinical Endocrinology and Metabolism

Volumn 87, Issue 2, 2002, Pages 951-

  Ozata, Metin ^a  

^a GULHANE SCHOOL OF MEDICINE   (Turkey)

Author keywords

[No Author keywords available]

Indexed keywords

LEPTIN;

BONE DENSITY; BONE MASS; BONE MINERAL; FEMALE; HUMAN; LETTER; MALE; MOUSE; NONHUMAN; OBESITY; OSSIFICATION; OSTEOLYSIS; OSTEOPENIA; PRIORITY JOURNAL; PROTEIN DEFICIENCY;

EID: 18244374490     PISSN: 0021972X     EISSN: None     Source Type: Journal    
DOI: 10.1210/jcem.87.2.8241     Document Type: Letter

References (10)

1. Editorial: The not-so-odd couple-The clinician and the experimentalist
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3. Effects of weight and body mass index on bone mineral density in mice and women: The Framingham study
4. Leptin and bone: Does the brain control bone biology?
5. Leptin acts on human marrow stromal precursor cells to enhance osteoblast differentiation and to inhibit adipocyte differentiation: A potential mechanism for increased bone mass in obesity
6. Leptin inhibits bone formation through a hypothalamic relay: A central control of bone mass
7. Leptin is a potent stimulator of bone growth in ob/ob mice
8. A leptin missense mutation associated with hypogonadism and morbid obesity
9. Human leptin deficiency caused by a missense mutation multiple endocrine defects, decreased sympathetic tone, and immune system dysfunction indicate new targets for leptin action, greater central than peripheral resistance to the effects of leptin, and spontaneous correction of leptin-mediated defects
10. Physiological roles of the leptin endocrine system: Differences between mice and humans