Co-exposure to pyridostigmine bromide, DEET, and/or permethrin causes sensorimotor deficit and alterations in brain acetylcholinesterase activity

Pharmacology Biochemistry and Behavior

Volumn 77, Issue 2, 2004, Pages 253-262

  Abou Donia, Mohamed B ^a   Dechkovskaia, Anjelika M ^a   Goldstein, Larry B ^a,b   Abdel Rahman, Ali ^a   Bullman, Sarah L ^a,b   Khan, Wasiuddin A ^a  

^a DUKE UNIVERSITY MEDICAL CENTER   (United States)

^b DURHAM VA MEDICAL CENTER   (United States)

Author keywords

Acetylcholinesterase; Brain; Butyrylcholinesterase; Combined exposure; DEET; M2 muscarinic acetylcholine receptor; Nicotinic acetylcholine receptor; Permethrin; Persian Gulf War; Pyridostigmine bromide; Sensorimotor

Indexed keywords

ACETYLCHOLINESTERASE; DIETHYLTOLUAMIDE; MUSCARINIC RECEPTOR; PERMETHRIN; PYRIDOSTIGMINE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN STEM; CEREBELLUM; COGNITION; CONTROLLED STUDY; ELECTROENCEPHALOGRAM; ENZYME ACTIVITY; EVALUATION; GRIP STRENGTH; LIGAND BINDING; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; SENSORIMOTOR FUNCTION; TASK PERFORMANCE;

ANIMALIA;

EID: 1642575181     PISSN: 00913057     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.pbb.2003.10.018     Document Type: Article

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