Ethanol withdrawal hyper-responsiveness mediated by NMDA receptors in spinal cord motor neurons

British Journal of Pharmacology

Volumn 139, Issue 1, 2003, Pages 73-80

  Li, Hui Fang ^a   Kendig, Joan J ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Ethanol; Motor neurons; NMDA; Protein kinase A; Protein kinase C; Spinal cord; Tyrosine kinase; Withdrawal

Indexed keywords

2 [1 (3 DIMETHYLAMINOPROPYL) 3 INDOLYL] 3 (3 INDOLYL)MALEIMIDE; ALCOHOL; ALPHA AMINO 3 HYDROXY 5 METHYL 4 ISOXAZOLEPROPIONIC ACID; CHELERYTHRINE; CYCLIC AMP DEPENDENT PROTEIN KINASE INHIBITOR; GENISTEIN; GLUTAMIC ACID; N METHYL DEXTRO ASPARTIC ACID; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; PROTEIN KINASE; PROTEIN KINASE C INHIBITOR; PROTEIN TYROSINE KINASE INHIBITOR;

ALCOHOL ABUSE; ALCOHOL WITHDRAWAL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVITY; EVOKED RESPONSE; EXCITATORY POSTSYNAPTIC POTENTIAL; NONHUMAN; PATCH CLAMP; PRIORITY JOURNAL; RAT; RECEPTOR INTRINSIC ACTIVITY; RECEPTOR UPREGULATION; SPINAL CORD MOTONEURON;

ALPHA-AMINO-3-HYDROXY-5-METHYL-4-ISOXAZOLEPROPIONIC ACID; ANIMALS; ETHANOL; EVOKED POTENTIALS, MOTOR; EXCITATORY AMINO ACID AGONISTS; GLUTAMIC ACID; MOTOR NEURONS; N-METHYLASPARTATE; PATCH-CLAMP TECHNIQUES; PROTEIN KINASE C; PROTEIN-SERINE-THREONINE KINASES; PROTEIN-TYROSINE KINASES; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, AMPA; RECEPTORS, N-METHYL-D-ASPARTATE; SPINAL CORD; SUBSTANCE WITHDRAWAL SYNDROME;

EID: 0038730724     PISSN: 00071188     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.bjp.0705198     Document Type: Article

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