Trichloroethylene decreases heat shock protein 90 interactions with endothelial nitric oxide synthase: Implications for endothelial cell proliferation

Toxicological Sciences

Volumn 73, Issue 1, 2003, Pages 90-97

  Ou, Jingsong ^a   Ou, Zhijun ^a   McCarver, D Gail ^a   Hines, Ronald N ^a   Oldham, Keith T ^a,b   Ackerman, Allan W ^a   Pritchard Jr , Kirkwood A ^a,b  

^a MEDICAL COLLEGE OF WISCONSIN   (United States)

^b FREE RADICAL RESEARCH CENTER   (United States)

Author keywords

Cardiac cells; Nitric oxide (NO); Septa; Superoxide anion(O2 ); Teratogens; Trichloroethylene (TRI)

Indexed keywords

CALCIMYCIN; CALCIUM IONOPHORE; HEAT SHOCK PROTEIN 90; N(G) NITROARGININE METHYL ESTER; NITRATE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITRITE; SERINE; SUPEROXIDE; TRICHLOROETHYLENE; VASCULOTROPIN;

ANGIOGENESIS; ANIMAL CELL; ARTICLE; CELL COUNT; CELL PROLIFERATION; CONGENITAL HEART MALFORMATION; CONTROLLED STUDY; DATA ANALYSIS; ENDOTHELIUM CELL; ENZYME ACTIVITY; HYPOTHESIS; IMMUNOBLOTTING; NONHUMAN; POLLUTANT; PROTEIN PROTEIN INTERACTION; STATISTICAL ANALYSIS;

ANIMALS; BLOTTING, WESTERN; CALCIMYCIN; CATTLE; CELL DIVISION; CELLS, CULTURED; ENDOTHELIAL CELLS; ENZYME INHIBITORS; HSP90 HEAT-SHOCK PROTEINS; IONOPHORES; NG-NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE III; PHOSPHORYLATION; PRECIPITIN TESTS; SUPEROXIDES; TRICHLOROETHYLENE; VASCULAR ENDOTHELIAL GROWTH FACTOR A;

ANIMALIA;

EID: 0038324417     PISSN: 10966080     EISSN: None     Source Type: Journal    
DOI: 10.1093/toxsci/kfg062     Document Type: Article

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