Oxidative stress caused by inactivation of glutathione peroxidase and adaptive responses

Biological Chemistry

Volumn 384, Issue 4, 2003, Pages 567-574

  Miyamoto, Yasuhide ^a   Koh, Young Ho ^a   Park, Yong Seek ^a   Fujiwara, Noriko ^a   Sakiyama, Haruhiko ^a   Misonou, Yoshiko ^a   Ookawara, Tomomi ^b   Suzuki, Keiichiro ^b   Honke, Koichi ^a   Taniguchi, Naoyuki ^a  

^a OSAKA UNIVERSITY MEDICAL SCHOOL   (Japan)

^b HYOGO COLLEGE OF MEDICINE   (Japan)

Author keywords

3 Deoxyglucosone; Aldehydes; Glutathione peroxidase; Methyl glyoxal; Nitric oxide; Peroxynitrite

Indexed keywords

ALDEHYDE DERIVATIVE; APOPTOSIS INHIBITOR; CARBONYL DERIVATIVE; DOXORUBICIN; FRUCTOSE; GLUTATHIONE PEROXIDASE; NITRIC OXIDE; NITROGEN OXIDE; PEROXIDE; THIOBARBITURIC ACID;

ANIMAL MODEL; APOPTOSIS; CARDIOMYOPATHY; CELL CULTURE; CELL DAMAGE; CELL FUNCTION; CELL PROTECTION; ENZYME ACTIVITY; ENZYME INACTIVATION; GENE EXPRESSION; HEART FAILURE; HUMAN; IN VITRO STUDY; NONHUMAN; OXIDATION REDUCTION STATE; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; SCAVENGING SYSTEM; SECOND MESSENGER; SHORT SURVEY;

ADAPTATION, PHYSIOLOGICAL; ALDEHYDES; ANIMALS; FRUCTOSE; GLUTATHIONE PEROXIDASE; HUMANS; NITRIC OXIDE; OXIDATIVE STRESS;

ANIMALIA;

EID: 0037621502     PISSN: 14316730     EISSN: None     Source Type: Journal    
DOI: 10.1515/BC.2003.064     Document Type: Short Survey

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