Inactivation of glutathione peroxidase by NO leads to the accumulation of H2O2 and the induction of HB-EGF via c-Jun NH2-terminal kinase in rat aortic smooth muscle cells.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 15, Issue 8, 2001, Pages 1472-1474

  Koh, Y H ^a   Suzuki, K ^a   Che, W ^a   Park, Y S ^a   Miyamoto, Y ^a   Higashiyama, S ^a   Taniguchi, N ^a  

^a OSAKA UNIVERSITY MEDICAL SCHOOL   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

EPIDERMAL GROWTH FACTOR; FAS ANTIGEN; GLUTATHIONE PEROXIDASE; HEPARIN BINDING EPIDERMAL GROWTH FACTOR; HEPARIN-BINDING EGF-LIKE GROWTH FACTOR; HYDROGEN PEROXIDE; MITOGEN ACTIVATED PROTEIN KINASE; NITRIC OXIDE; STRESS ACTIVATED PROTEIN KINASE;

ANIMAL; AORTA; ARTICLE; BIOLOGICAL MODEL; BIOSYNTHESIS; CYTOLOGY; DRUG ANTAGONISM; DRUG EFFECT; GENE EXPRESSION REGULATION; METABOLISM; RAT; SIGNAL TRANSDUCTION; VASCULAR SMOOTH MUSCLE;

ANIMALS; ANTIGENS, CD95; AORTA; EPIDERMAL GROWTH FACTOR; GENE EXPRESSION REGULATION, ENZYMOLOGIC; GLUTATHIONE PEROXIDASE; HYDROGEN PEROXIDE; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MITOGEN-ACTIVATED PROTEIN KINASES; MODELS, BIOLOGICAL; MUSCLE, SMOOTH, VASCULAR; NITRIC OXIDE; RATS; SIGNAL TRANSDUCTION;

EID: 0035377253     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.00-0572fje     Document Type: Article

References (0)