Chronic inhibition of Rho kinase blunts the process of left ventricular hypertrophy leading to cardiac contractile dysfunction in hypertension-induced heart failure

Journal of Molecular and Cellular Cardiology

Volumn 35, Issue 1, 2003, Pages 59-70

  Satoh, Shinji ^a   Ueda, Yasuko ^a   Koyanagi, Masamichi ^a   Kadokami, Toshiaki ^a   Sugano, Masahiro ^a   Yoshikawa, Yasuji ^a   Makino, Naoki ^a  

^a KYUSHU UNIVERSITY   (Japan)

Author keywords

Cardiac contraction; G proteins; Heart failure; Myocardial hypertrophy; Rho kinase

Indexed keywords

4 (1 AMINOETHYL) N (4 PYRIDYL)CYCLOHEXANECARBOXAMIDE; PROTEIN KINASE INHIBITOR; RHO KINASE; SODIUM CHLORIDE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; HEART FAILURE; HEART LEFT VENTRICLE DP-DT; HEART LEFT VENTRICLE EJECTION FRACTION; HEART LEFT VENTRICLE HYPERTROPHY; HEART LEFT VENTRICLE MASS; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILE FORCE; HEART MUSCLE CONTRACTILITY; HEART MUSCLE FIBER MEMBRANE; HEART MUSCLE FIBROSIS; HEART MUSCLE RELAXATION; HEART PAPILLARY MUSCLE; HYPERTENSION; MUSCLE TWITCH; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT;

AMIDES; ENZYME INHIBITORS; GTP-BINDING PROTEIN ALPHA SUBUNITS, GQ-G11; HEART FAILURE, CONGESTIVE; HETEROTRIMERIC GTP-BINDING PROTEINS; HYPERTENSION; HYPERTROPHY, LEFT VENTRICULAR; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MYOCARDIUM; PROTEIN-SERINE-THREONINE KINASES; PYRIDINES;

EID: 0037214686     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0022-2828(02)00278-X     Document Type: Article

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