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Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 2, 2002, Pages 943-948

Polycystic kidneys and chronic inflammatory lesions are the delayed consequences of loss of the suppressor of cytokine signaling-1 (SOCS-1)

(6) Metcalf, Donald a Mifsud, Sandra a Di Rago, Ladina a Nicola, Nicos A a Hilton, Douglas J a Alexander, Warren S a

a ROYAL MELBOURNE HOSPITAL (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA INTERFERON; SUPPRESSOR FACTOR; SUPPRESSOR OF CYTOKINE SIGNALING 1; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CATARACT; CHRONIC INFLAMMATION; CONTROLLED STUDY; DISEASE COURSE; FEMALE; GENE DELETION; GENE INACTIVATION; GRANULOMA; KIDNEY POLYCYSTIC DISEASE; KIDNEY TUBULE; LIVER FATTY DEGENERATION; MALE; MORTALITY; MOUSE; NEWBORN; NONHUMAN; PNEUMONIA; PRIORITY JOURNAL; SKIN ULCER; T CELL LEUKEMIA;

ANIMALS; ANIMALS, NEWBORN; CARRIER PROTEINS; FATTY LIVER; FEMALE; INFLAMMATION; INTERFERON TYPE II; LEUKEMIA, LYMPHOCYTIC; LONGEVITY; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; PHENOTYPE; POLYCYSTIC KIDNEY DISEASES; REPRESSOR PROTEINS; SUPPRESSOR OF CYTOKINE SIGNALING PROTEINS;

ANIMALIA;

EID: 0037154210 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.022628499 Document Type: Article

Times cited : (83)

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