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Volumn 297, Issue 5589, 2002, Pages 2066-2070
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Evolution of autoantibody responses via somatic hypermutation outside of germinal centers
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Author keywords
[No Author keywords available]
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Indexed keywords
CELLS;
DISEASES;
HYPERMUTATION;
ANTIBODIES;
AUTOANTIBODY;
FAS ANTIBODY;
IMMUNOGLOBULIN G2A;
MONOCLONAL ANTIBODY;
RHEUMATOID FACTOR;
ANTIBODY;
IMMUNE RESPONSE;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ANTIBODY PRODUCTION;
ANTIBODY RESPONSE;
ARTICLE;
AUTOIMMUNE DISEASE;
B LYMPHOCYTE;
CONTROLLED STUDY;
GERMINAL CENTER;
IMMUNOHISTOCHEMISTRY;
IMMUNOLOGICAL TOLERANCE;
LYMPHOCYTE PROLIFERATION;
MOUSE;
NONHUMAN;
POLYMERASE CHAIN REACTION;
PRIORITY JOURNAL;
SOMATIC MUTATION;
SPLEEN CELL;
AMINO ACID SEQUENCE;
ANIMALS;
ANTIBODIES, ANTI-IDIOTYPIC;
AUTOANTIBODIES;
AUTOIMMUNE DISEASES;
AUTOIMMUNITY;
B-LYMPHOCYTES;
BASE SEQUENCE;
DENDRITIC CELLS, FOLLICULAR;
GENES, IMMUNOGLOBULIN;
GERMINAL CENTER;
IMMUNOGLOBULIN VARIABLE REGION;
MICE;
MICE, INBRED MRL LPR;
MICE, TRANSGENIC;
MOLECULAR SEQUENCE DATA;
RHEUMATOID FACTOR;
SELF TOLERANCE;
SOMATIC HYPERMUTATION, IMMUNOGLOBULIN;
SPLEEN;
T-LYMPHOCYTES;
ANIMALIA;
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EID: 0037144519
PISSN: 00368075
EISSN: None
Source Type: Journal
DOI: 10.1126/science.1073924 Document Type: Article |
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Times cited : (443)
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References (26)
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