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Volumn 446, Issue 1-3, 2002, Pages 53-61
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Amobarbital inhibits K+-stimulated glucose oxidation in cerebellar granule neurons by two mechanisms
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Author keywords
Anesthesia; Barbiturates; Cytoprotection; Glucose phosphorylation; Glutamate; Oxidative metabolism
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Indexed keywords
AMOBARBITAL;
GLUCOSE;
GLUTAMIC ACID;
HYPNOTIC SEDATIVE AGENT;
LACTIC ACID;
POTASSIUM;
CARBON DIOXIDE;
POTASSIUM ION;
ANIMAL;
ARTICLE;
BIOSYNTHESIS;
CELL CULTURE;
CEREBELLUM;
CHEMICAL PHENOMENA;
CYTOLOGY;
DRUG EFFECT;
ENERGY METABOLISM;
GLYCOLYSIS;
KINETICS;
METABOLISM;
MOUSE;
NERVE CELL;
OXIDATION REDUCTION REACTION;
ANIMAL CELL;
BRAIN NERVE CELL;
CONCENTRATION RESPONSE;
CONTROLLED STUDY;
DRUG MECHANISM;
DRUG POTENCY;
GLUCOSE OXIDATION;
MITOCHONDRIAL RESPIRATION;
NONHUMAN;
PRIORITY JOURNAL;
AMOBARBITAL;
ANIMALS;
CELLS, CULTURED;
CEREBELLUM;
DEPRESSION, CHEMICAL;
ENERGY METABOLISM;
GLUCOSE;
GLUTAMIC ACID;
GLYCOLYSIS;
HYPNOTICS AND SEDATIVES;
KINETICS;
LACTIC ACID;
MICE;
NEURONS;
OXIDATION-REDUCTION;
POTASSIUM;
ANIMAL;
SEDATIVES, BARBITURATE;
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EID: 0037141758
PISSN: 00142999
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-2999(02)01794-6 Document Type: Article |
Times cited : (9)
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References (74)
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