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Volumn 33, Issue 1, 2002, Pages 261-267
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Inhibition of glutamate release via recovery of ATP levels accounts for a neuroprotective effect of aspirin in rat cortical neurons exposed to oxygen-glucose deprivation
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Author keywords
Aspirin; Cell respiration; Glutamates; Ischemia; Mitochondria; Neuronal death; Neurons; Salicylates; Stroke, experimental
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Indexed keywords
ACETYLSALICYLATE COPPER;
ADENOSINE TRIPHOSPHATE;
ETHYLENE GLYCOL 1,2 BIS(2 AMINOPHENYL) ETHER N,N,N',N' TETRAACETIC ACID;
GLUTAMATE TRANSPORTER;
GLUTAMIC ACID;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
INDOMETACIN;
LACTATE DEHYDROGENASE;
PROSTAGLANDIN SYNTHASE;
PYRROLIDINE DITHIOCARBAMATE;
SALICYLATE SODIUM;
ANIMAL CELL;
ARTICLE;
BRAIN CORTEX;
BRAIN MITOCHONDRION;
BRAIN OXYGEN CONSUMPTION;
CEREBROVASCULAR ACCIDENT;
CONTROLLED STUDY;
DRUG MECHANISM;
FETUS;
GLIA CELL;
NERVE CELL NECROSIS;
NEUROPROTECTION;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
ADENOSINE TRIPHOSPHATE;
ANIMALS;
ASPIRIN;
BIOLOGICAL TRANSPORT;
BRAIN;
BRAIN ISCHEMIA;
CELL DEATH;
CELL HYPOXIA;
CELL RESPIRATION;
CELLS, CULTURED;
GLUCOSE;
GLUTAMIC ACID;
MITOCHONDRIA;
NEURONS;
NEUROPROTECTIVE AGENTS;
OXYGEN CONSUMPTION;
RATS;
RATS, WISTAR;
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EID: 0036141158
PISSN: 00392499
EISSN: None
Source Type: Journal
DOI: 10.1161/hs0102.101299 Document Type: Article |
Times cited : (93)
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References (27)
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