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Volumn 33, Issue 1, 2002, Pages 261-267

Inhibition of glutamate release via recovery of ATP levels accounts for a neuroprotective effect of aspirin in rat cortical neurons exposed to oxygen-glucose deprivation

Author keywords

Aspirin; Cell respiration; Glutamates; Ischemia; Mitochondria; Neuronal death; Neurons; Salicylates; Stroke, experimental

Indexed keywords

ACETYLSALICYLATE COPPER; ADENOSINE TRIPHOSPHATE; ETHYLENE GLYCOL 1,2 BIS(2 AMINOPHENYL) ETHER N,N,N',N' TETRAACETIC ACID; GLUTAMATE TRANSPORTER; GLUTAMIC ACID; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INDOMETACIN; LACTATE DEHYDROGENASE; PROSTAGLANDIN SYNTHASE; PYRROLIDINE DITHIOCARBAMATE; SALICYLATE SODIUM;

EID: 0036141158     PISSN: 00392499     EISSN: None     Source Type: Journal    
DOI: 10.1161/hs0102.101299     Document Type: Article
Times cited : (93)

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  • 27
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    • Adenosine triphosphate depletion reverses sodium-dependent, neuronal uptake of glutamate in rat hippocampal slices
    • (1993) J Neurosci , vol.13 , pp. 4429-4444
    • Madl, J.E.1    Burgesser, K.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.