Cell transformation by v-Jun deactivates ERK MAP kinase signalling

Oncogene

Volumn 21, Issue 42, 2002, Pages 6540-6548

  Black, Elizabeth J ^a   Mark, Walker ^a   Clark, William ^a   MacLaren, Ann ^a   Gillespie, David A F ^b  

^a BEATSON INSTITUTE FOR CANCER RESEARCH   (United Kingdom)

^b UNIVERSITY OF GLASGOW   (United Kingdom)

Author keywords

Autocrine; MAP kinase; Transformation; v Jun

Indexed keywords

EPIDERMAL GROWTH FACTOR; MITOGEN ACTIVATED PROTEIN KINASE; ONCOPROTEIN; PHORBOL ESTER; PROTEIN V JUN; RAF PROTEIN; RAS PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; CARCINOGENESIS; CELL CYCLE G1 PHASE; CELL CYCLE S PHASE; CELL FUNCTION; CELL TRANSFORMATION; CONTROLLED STUDY; EMBRYO; ENZYME INACTIVATION; ENZYME PHOSPHORYLATION; ENZYME REGULATION; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ANIMALS; BLOTTING, WESTERN; CELL DIVISION; CELL TRANSFORMATION, NEOPLASTIC; CHICK EMBRYO; DOWN-REGULATION; FIBROBLASTS; HUMANS; IMMUNOENZYME TECHNIQUES; MAP KINASE KINASE KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASES; ONCOGENE PROTEIN P65(GAG-JUN); PHOSPHORYLATION; PROTEIN-SERINE-THREONINE KINASES; RETROVIRIDAE; SIGNAL TRANSDUCTION; TETRADECANOYLPHORBOL ACETATE; TUMOR CELLS, CULTURED;

ANIMALIA;

EID: 0037136696     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.onc.1205851     Document Type: Article

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