Disruption of inducible nitric oxide synthase improves β-adrenergic inotropic responsiveness but not the survival of mice with cytokine-induced cardiomyopathy

Circulation Research

Volumn 90, Issue 9, 2002, Pages 959-965

  Funakoshi, Hajime ^a   Kubota, Toru ^a   Kawamura, Natsumi ^a   Machida, Yoji ^a   Feldman, Arthur M ^a   Tsutsui, Hiroyuki ^a   Shimokawa, Hiroaki ^a   Takeshita, Akira ^a  

^a KYUSHU UNIVERSITY   (Japan)

Author keywords

Cytokines; Heart failure; Nitric oxide synthase; Transgenic mice; Tumor necrosis factor

Indexed keywords

BETA ADRENERGIC RECEPTOR; CYTOKINE; NITRIC OXIDE SYNTHASE; TUMOR NECROSIS FACTOR ALPHA;

AGE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CARDIOMYOPATHY; CONTROLLED STUDY; ENZYME INHIBITION; GENE DISRUPTION; GENE OVEREXPRESSION; HEART LEFT VENTRICLE PRESSURE; HEART MUSCLE; HEART MUSCLE CONTRACTILITY; HEART VENTRICLE HYPERTROPHY; INFLAMMATION; INOTROPISM; MOUSE; MYOCARDITIS; NONHUMAN; PRIORITY JOURNAL; RISK FACTOR; SEX DIFFERENCE; SURVIVAL;

ADRENERGIC BETA-AGONISTS; ANIMALS; BLOOD PRESSURE; BLOTTING, WESTERN; CARDIOMYOPATHIES; ENZYME INDUCTION; FEMALE; GENOTYPE; HEART RATE; ISOPROTERENOL; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, TRANSGENIC; MUTATION; MYOCARDIAL CONTRACTION; MYOCARDIUM; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE II; NITRIC OXIDE SYNTHASE TYPE III; ORGAN SIZE; SURVIVAL ANALYSIS; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0037124005     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.RES.0000017632.83720.68     Document Type: Article

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