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Volumn 21, Issue 22, 2002, Pages 3611-3619

Enhanced tumor killing by Apo2L/TRAIL and CPT-11 co-treatment is associated with p21 cleavage and differential regulation of Apo2L/TRAIL ligand and its receptors

(8) Xiang, Hong a Fox, Judith A a Totpal, Klara a Aikawa, Mina a Dupree, Kelly a Sinicropi, Dominick a Lowe, John a Escandón, Enrique a

a GENENTECH INC (United States)

Author keywords

Apo2L TRAIL; CPT 11; DR4; DR5; p21; SN 38

Indexed keywords

CASPASE; IRINOTECAN; MESSENGER RNA; PROTEIN; PROTEIN P21; PROTEIN P53; RECEPTOR; TUMOR NECROSIS FACTOR RELATED APOPTOSIS INDUCING LIGAND;

APOPTOSIS; ARTICLE; CONTROLLED STUDY; ENZYME ACTIVATION; HUMAN; HUMAN CELL; IMMUNOBLOTTING; PRIORITY JOURNAL; PROTEIN DEGRADATION; REGULATORY MECHANISM; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; TUMOR;

ANTINEOPLASTIC AGENTS, PHYTOGENIC; APOPTOSIS; APOPTOSIS REGULATORY PROTEINS; CAMPTOTHECIN; CARCINOMA; CASPASES; CELL CYCLE; CELLS, CULTURED; COLONIC NEOPLASMS; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DRUG SYNERGISM; HUMANS; KINETICS; MEMBRANE GLYCOPROTEINS; NEOPLASMS; RECEPTORS, TUMOR NECROSIS FACTOR; RNA, NEOPLASM; TNF-RELATED APOPTOSIS-INDUCING LIGAND; TUMOR CELLS, CULTURED; TUMOR NECROSIS FACTOR-ALPHA;

ANIMALIA;

EID: 0037118590 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205449 Document Type: Article

Times cited : (54)

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