WT1 is a key regulator of podocyte function: Reduced expression levels cause crescentic glomerulonephritis and mesangial sclerosis

Human Molecular Genetics

Volumn 11, Issue 6, 2002, Pages 651-659

  Guo, Jian Kan ^a,f   Menke, Aswin L ^b   Gubler, Marie Claire ^c   Clarke, Alan R ^d   Harrison, David ^e   Hammes, Annette ^a   Hastie, Nicholas D ^b   Schedl, Andreas ^a,f  

^a MAX DELBRÜCK CENTER FOR MOLECULAR MEDICINE   (Germany)

^b UNIVERSITY OF EDINBURGH   (United Kingdom)

^c HÔPITAL NECKER ENFANTS MALADES   (France)

^d CARDIFF UNIVERSITY   (United Kingdom)

^e UNIVERSITY OF EDINBURGH   (United Kingdom)

^f NEWCASTLE UNIVERSITY   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

NEPHRIN; PODOCALYXIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR WT1; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL FUNCTION; CONTROLLED STUDY; DATA ANALYSIS; GENE CONTROL; GENE DOSAGE; GENE EXPRESSION; GLOMERULONEPHRITIS; GLOMERULOSCLEROSIS; KIDNEY DISEASE; KNOCKOUT GENE; MESANGIUM CELL; MOUSE; NONHUMAN; PATHOGENESIS; PODOCYTE; PRIORITY JOURNAL; TRANSGENIC MOUSE; TUMOR SUPPRESSOR GENE; WAGR SYNDROME; YEAST ARTIFICIAL CHROMOSOME;

ANIMALIA; MUS MUSCULUS;

EID: 0037087593     PISSN: 09646906     EISSN: None     Source Type: Journal    
DOI: 10.1093/hmg/11.6.651     Document Type: Article

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