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Oncogene

Volumn 21, Issue 26, 2002, Pages 4176-4180

Variant XRCC3 implicated in cancer is functional in homology-directed repair of double-strand breaks

(4) Araujo, Felipe D a Pierce, Andrew J a Stark, Jeremy M a Jasin, Maria a,b

a MEMORIAL SLOAN KETTERING CANCER CENTER (United States)

b Weill Cornell Graduate School of Biomedical Sciences (United States)

Author keywords

DNA double strand break repair; Homologous recombination; Polymorphism; RAD51; XRCC3

Indexed keywords

GENE PRODUCT; MITOMYCIN C; PROTEIN XRCC3; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; CANCER GENETICS; CANCER RISK; CONTROLLED STUDY; CROSS LINKING; DNA REPAIR; EVOLUTIONARY HOMOLOGY; FLUORESCENCE; GENE EXPRESSION; GENE MUTATION; GENETIC COMPLEMENTATION; GENETIC POLYMORPHISM; GENETIC VARIABILITY; NONHUMAN; PRIORITY JOURNAL; RISK ASSESSMENT;

ANIMALIA;

EID: 0037071901 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205539 Document Type: Article

Times cited : (100)

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