Unique topographical distribution of M cells underlies reentrant mechanism of torsade de pointes in the long-QT syndrome

Circulation

Volumn 105, Issue 10, 2002, Pages 1247-1253

  Akar, Fadi G ^a   Yan, Gan Xin ^b   Antzelevitch, Charles ^b   Rosenbaum, David S ^a,c  

^a Case Western Reserve University   (United States)

^b MASONIC MEDICAL RESEARCH LABORATORY   (United States)

^c CASE WESTERN RESERVE UNIVERSITY   (United States)

Author keywords

Action potentials; Arrhythmia; Ion channels; Mapping; Torsades de pointes

Indexed keywords

ION CHANNEL;

ACTION POTENTIAL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARRHYTHMOGENESIS; ARTICLE; DOG; ELECTROPHYSIOLOGY; HEART MUSCLE CELL; HEART REPOLARIZATION; LONG QT SYNDROME; NONHUMAN; PRIORITY JOURNAL; TORSADE DES POINTES;

ACTION POTENTIALS; ANIMALS; BODY SURFACE POTENTIAL MAPPING; DISEASE MODELS, ANIMAL; DOGS; ELECTRIC STIMULATION; ELECTROCARDIOGRAPHY; ELECTROPHYSIOLOGIC TECHNIQUES, CARDIAC; HEART CONDUCTION SYSTEM; LONG QT SYNDROME; MYOCARDIUM; SOTALOL; TORSADES DE POINTES;

EID: 0037066015     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/hc1002.105231     Document Type: Article

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