The preferential ability of B lymphocytes to act as diabetogenic APC in NOD mice depends on expression of self-antigen-specific immunoglobulin receptors

European Journal of Immunology

Volumn 32, Issue 12, 2002, Pages 3657-3666

  Silveira, Pablo A ^a   Johnson, Ellis ^a   Chapman, Harold D ^a   Bui, Thi ^b   Tisch, Roland M ^b   Serreze, David V ^a  

^a JACKSON LABORATORY   (United States)

^b UNIVERSITY OF NORTH CAROLINA SCHOOL OF MEDICINE   (United States)

Author keywords

Antigen presentation; Auto antibody; B lymphocyte; NOD mice; Processing; Type 1 diabetes

Indexed keywords

AUTOANTIGEN; CD4 ANTIGEN; CELL PROTEIN; GLUTAMATE DECARBOXYLASE; IMMUNOGLOBULIN; IMMUNOGLOBULIN MU CHAIN; IMMUNOGLOBULIN RECEPTOR; LYSOZYME;

ALLELE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIGEN PRESENTING CELL; ANTIGEN SPECIFICITY; ARTICLE; AUTOIMMUNE DISEASE; B LYMPHOCYTE; CONTROLLED STUDY; DIABETOGENESIS; FEMALE; GENE EXPRESSION; GENE MUTATION; IMMUNOLOGICAL TOLERANCE; INSULIN DEPENDENT DIABETES MELLITUS; LYMPHOCYTE FUNCTION; MALE; MOUSE; MOUSE STRAIN; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; T LYMPHOCYTE; TRANSGENE;

EID: 0036918641     PISSN: 00142980     EISSN: None     Source Type: Journal    
DOI: 10.1002/1521-4141(200212)32:12<3657::AID-IMMU3657>3.0.CO;2-E     Document Type: Article

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