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Volumn 38, Issue 2, 2002, Pages 131-136

Inactivation of p16INK4a expression in malignant mesothelioma by methylation

Author keywords

cdk; Cell cycle; Epigenetic

Indexed keywords

ARTICLE; CELL CYCLE; CELL GROWTH; CELL LINE; CONTROLLED STUDY; DNA METHYLATION; EXON; GENE EXPRESSION REGULATION; HUMAN; HUMAN CELL; LUNG CANCER; MESOTHELIOMA; PRIORITY JOURNAL; BIOSYNTHESIS; CELL CULTURE; GENETICS; PATHOPHYSIOLOGY; POLYMERASE CHAIN REACTION;

EID: 0036837992     PISSN: 01695002     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0169-5002(02)00178-2     Document Type: Article
Times cited : (80)

References (35)
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    • Herman, J.G.1    Merlo, A.2    Mao, L.3
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    • 0032569736 scopus 로고    scopus 로고
    • Expression of p16INK4a/p16alpha and p19ARF/p16beta is frequently altered in non-small cell lung cancer and correlates with p53 overexpression
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    • Vonlanthen, S.1    Heighway, J.2    Tschan, M.P.3
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    • Daskalakis M, Nguyen TT, Wijermans P, Jones PA, Lubbert M. Frequent hypermethylation of p15/INK4b in patients with myelodysplastic syndromes is decreased following treatment with 5-aza-2′-deoxycytidine (Decitabine). In: American Society of Hematology 40th annual meeting, Miami Beach, FL, 1998
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.