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Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 16, 2002, Pages 10635-10640

A human embryonic hemoglobin inhibits Hb S polymerization in vitro and restores a normal phenotype to mouse models of sickle cell disease

(2) He, Zhenning a Russell, J Eric a

a UNIVERSITY OF PENNSYLVANIA (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BETA GLOBIN; GAMMA GLOBIN; HEMOGLOBIN DERIVATIVE; HEMOGLOBIN F; HEMOGLOBIN GOWER 2; HEMOGLOBIN S; UNCLASSIFIED DRUG;

ANIMAL CELL; ARTICLE; CLINICAL FEATURE; CONTROLLED STUDY; DEVELOPMENTAL STAGE; EXTRAMEDULLARY HEMATOPOIESIS; GENE ACTIVATION; GENE EXPRESSION; GENE SILENCING; GLOBIN GENE; HEMOLYTIC ANEMIA; HISTOPATHOLOGY; IN VITRO STUDY; IN VIVO STUDY; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN POLYMERIZATION; SICKLE CELL ANEMIA; TISSUE INJURY;

ANEMIA, SICKLE CELL; ANIMALS; BETA-THALASSEMIA; DISEASE MODELS, ANIMAL; ERYTHROCYTES; GENE EXPRESSION; GLOBINS; HEMATOPOIESIS, EXTRAMEDULLARY; HEMOGLOBIN SC DISEASE; HEMOGLOBIN, SICKLE; HEMOGLOBINS, ABNORMAL; HUMANS; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; PHENOTYPE; POLYMERS; SYNDROME;

ANIMALIA;

EID: 0036678156 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.162269099 Document Type: Article

Times cited : (16)

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