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Volumn 188, Issue 1, 2002, Pages 73-86
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External pore collapse as an inactivation mechanism for Kv4.3 K+ channels
a a a a a |
Author keywords
C inactivation; Channel inactivation; Inactivation; Ito; K channel; Kv4; Kv4.3
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Indexed keywords
POTASSIUM CHANNEL;
DELAYED RECTIFIER POTASSIUM CHANNEL;
KCND3 PROTEIN, HUMAN;
POTASSIUM;
POTASSIUM CHANNEL BLOCKING AGENT;
SHAL POTASSIUM CHANNEL;
VOLTAGE GATED POTASSIUM CHANNEL;
AMINO TERMINAL SEQUENCE;
ARTICLE;
BINDING AFFINITY;
BINDING SITE;
CARBOXY TERMINAL SEQUENCE;
CHANNEL GATING;
CONFORMATIONAL TRANSITION;
CYTOPLASM;
HUMAN;
HUMAN CELL;
POTASSIUM CONDUCTANCE;
REGULATORY MECHANISM;
BIOLOGICAL MODEL;
CELL LINE;
CELL MEMBRANE POTENTIAL;
DRUG EFFECT;
ELECTRIC CONDUCTIVITY;
KIDNEY;
METABOLISM;
PATCH CLAMP;
PHYSIOLOGY;
PRENATAL DEVELOPMENT;
CELL LINE;
ELECTRIC CONDUCTIVITY;
HUMAN;
ION CHANNEL GATING;
KIDNEY;
MEMBRANE POTENTIALS;
MODELS, BIOLOGICAL;
PATCH-CLAMP TECHNIQUES;
POTASSIUM;
POTASSIUM CHANNEL BLOCKERS;
POTASSIUM CHANNELS;
SUPPORT, NON-U.S. GOV'T;
SUPPORT, U.S. GOV'T, P.H.S.;
DELAYED RECTIFIER POTASSIUM CHANNELS;
HUMANS;
POTASSIUM CHANNELS, VOLTAGE-GATED;
SHAL POTASSIUM CHANNELS;
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EID: 0036628856
PISSN: 00222631
EISSN: None
Source Type: Journal
DOI: 10.1007/s00232-001-0173-3 Document Type: Article |
Times cited : (25)
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References (40)
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