In vitro motility analysis of thin filaments from failing and non-failing human heart: Troponin from failing human hearts induces slower filament sliding and higher Ca2+ sensitivity

Journal of Molecular and Cellular Cardiology

Volumn 34, Issue 4, 2002, Pages 469-482

  Knott, Adam ^a   Purcell, Ian ^a   Marston, Steven ^a  

^a NATIONAL HEART AND LUNG INSTITUTE   (United Kingdom)

Author keywords

Ca2+ regulation; Heart failure; In vitro motility; Phosphorylation; Troponin

Indexed keywords

ACTIN; CALCIUM ION; MYOSIN HEAVY CHAIN; TROPOMYOSIN; TROPONIN I; TROPONIN T; CALCIUM; ISOPROTEIN; PHOSPHATASE; PHOSPHOTRANSFERASE; TROPONIN;

ADOLESCENT; ADULT; ARTICLE; CONTROLLED STUDY; FEMALE; FETUS; HEART FAILURE; HUMAN; HUMAN TISSUE; INFANT; MALE; MUSCLE CONTRACTILITY; NONHUMAN; NORMAL HUMAN; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DEPHOSPHORYLATION; PROTEIN EXPRESSION; PROTEIN PHOSPHORYLATION; THIN FILAMENT; VELOCITY; WESTERN BLOTTING; CONGESTIVE HEART FAILURE; HEART CONTRACTION; HEART MUSCLE; IN VITRO STUDY; METABOLISM; MICROFILAMENT; PHOSPHORYLATION;

CALCIUM; HEART FAILURE, CONGESTIVE; HUMANS; MICROFILAMENTS; MYOCARDIAL CONTRACTION; MYOCARDIUM; PHOSPHORIC MONOESTER HYDROLASES; PHOSPHORYLATION; PHOSPHOTRANSFERASES; PROTEIN ISOFORMS; TROPOMYOSIN; TROPONIN;

EID: 0036547621     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1006/jmcc.2002.1528     Document Type: Article

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